Muhyieddeen Amer Hussein, Asadourian Miro, Kunchakarra Siri, Rathod Ankit
Internal Medicine, UCSF Fresno Center for Medical Education and Research Edward and Ann Hildebrand Medical Library, Fresno, California, USA
Internal Medicine, UCSF Fresno Center for Medical Education and Research Edward and Ann Hildebrand Medical Library, Fresno, California, USA.
BMJ Case Rep. 2021 Mar 19;14(3):e240626. doi: 10.1136/bcr-2020-240626.
Lipomatous metaplasia in chronic postmyocardial infarction scars is a common and underappreciated finding seen in histopathology and cardiac MRI. Evidence suggests that lipomatous metaplasia is capable of altering the electroconductivity of the myocardium leading to re-entry pathways that are implicated in the pathogenesis of postmyocardial infarction arrhythmogenesis. We report a case of a patient who presented with non-sustained ventricular tachycardia and was found to have lipomatous metaplasia of a prior myocardial infarct-related scar.
慢性心肌梗死后瘢痕中的脂肪化生是组织病理学和心脏磁共振成像中常见但未得到充分认识的发现。有证据表明,脂肪化生能够改变心肌的电导率,导致折返通路,这与心肌梗死后心律失常的发病机制有关。我们报告一例患者,该患者出现非持续性室性心动过速,经检查发现既往心肌梗死相关瘢痕存在脂肪化生。