Ly6c 浸润巨噬细胞促进损伤条件性小鼠的囊泡进展。
Ly6c Infiltrating Macrophages Promote Cyst Progression in Injured Conditional Mice.
机构信息
Department of Cell, Developmental, and Integrative Biology, University of Alabama at Birmingham, Birmingham, Alabama.
Department of Medicine, Division of Nephrology, University of Alabama at Birmingham, Birmingham, Alabama.
出版信息
Kidney360. 2021 Jun 24;2(6):989-995. doi: 10.34067/KID.0000882021.
Abstract
Ly6c infiltrating macrophage numbers are increased in injured, conditional mice, compared with controls. Loss of Ly6c infiltrating macrophages slows injury-accelerated cystic disease. Ly6c infiltrating macrophages drive cystic disease in non-–deficient cystic models.
摘要
与对照相比，损伤条件性小鼠中 Ly6c 浸润巨噬细胞数量增加。缺失 Ly6c 浸润巨噬细胞会减缓损伤加速的囊状疾病。Ly6c 浸润巨噬细胞驱动非缺陷性囊状模型中的囊状疾病。
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