Anesthesiology and Pain Medicine, University of Washington School of Medicine. Anesthesiologist, Seattle Children's Hospital, WA.
Clinical Professor in the Department of Anesthesia and Pain Medicine at University of California, Irvine, CA.
Pain Physician. 2022 Jul;25(4):E503-E521.
The superior and middle cluneal nerves are sources of low back, buttock, and leg pain. These nerves are cutaneous branches of the lateral branches of the dorsal rami of T11- S4. Pain arising from entrapment or dysfunction of one or more of these nerves is called "cluneal nerve syndrome." A clear understanding of the anatomy underlying cluneal nerve syndrome and its treatment has been hampered by the very small size of the cluneal nerves and their complex, varying anatomy. Because of differing methods and foci of investigation, the literature regarding cluneal nerves has been confusing and even contradictory.
This paper provides a thorough critical literature review of cluneal nerve anatomy and implications for therapy.
A modified scoping review.
The bibliographic trail of English language papers on the anatomy and treatment of cluneal nerve syndrome was used to resolve the contradictions that have appeared in some of the anatomic descriptions and, where applicable, to examine their implications for therapy.
Recent anatomic and surgical investigations confirm a wider than previously realized range of central nervous system origins of these peripheral nerves, explaining why cluneal nerve dysfunction can cause a wide array of symptoms, including low back, buttock, and/or leg pain or "pseudosciatica."
Cluneal nerve syndrome is characterized by a triad of pain, tender points, and relief with local anesthetic injections. The pain is a deep, aching, poorly localized low back pain with variable involvement of the buttocks and/or legs. Tender points are localized at the iliac crest or caudal to the posterior superior iliac spine. Muscle weakness and dermatomal sensory changes are absent in cluneal nerve syndrome. If the pain returns after injections, neuroablation, nerve stimulation, or surgical release may be needed.
臀上和臀中皮神经是引起腰、臀和下肢疼痛的根源。这些神经是 T11-S4 背侧支的外侧支的皮支。由这些神经的一处或多处受压或功能障碍引起的疼痛称为“臀上皮神经综合征”。由于臀上皮神经非常小,其解剖结构复杂且多变,对其发病机制及其治疗的理解一直受到限制。由于研究方法和关注点不同,有关臀上皮神经的文献一直令人困惑,甚至相互矛盾。
本文对臀上皮神经解剖及其治疗意义进行了全面的批判性文献回顾。
改良的范围综述。
利用英语文献臀上皮神经综合征解剖学和治疗的文献轨迹,解决了一些解剖描述中出现的矛盾,并在适用的情况下,对其治疗意义进行了检查。
最近的解剖学和外科研究证实,这些周围神经的中枢神经系统起源范围比以前认识的要广泛,这解释了为什么臀上皮神经功能障碍会引起广泛的症状,包括腰、臀和/或下肢疼痛或“假性坐骨神经痛”。
臀上皮神经综合征的特征是疼痛、压痛和局部麻醉注射缓解三联征。疼痛为深部、隐痛、定位不清的腰痛,臀部和/或下肢受累程度不一。压痛位于髂嵴或后上髂嵴下方。臀上皮神经综合征无肌肉无力和皮节感觉改变。如果注射后疼痛复发,可能需要神经消融、神经刺激或手术松解。