勘误：长链非编码RNA表达增加调节香烟烟雾和慢性阻塞性肺疾病相关的气道炎症及黏液细胞增生。
Corrigendum: Increased expression of LncRNA regulates the cigarette smoke and COPD associated airway inflammation and mucous cell hyperplasia.
作者信息
Manevski Marko, Devadoss Dinesh, Long Christopher, Singh Shashi P, Nasser Mohd Wasim, Borchert Glen M, Nair Madhavan N, Rahman Irfan, Sopori Mohan, Chand Hitendra S
机构信息
Department of Immunology and Nano-Medicine, Herbert Wertheim College of Medicine, Florida International University, Miami, FL, United States.
Respiratory Immunology Program, Lovelace Respiratory Research Institute, Albuquerque, NM, United States.
出版信息
Front Immunol. 2022 Aug 8;13:988069. doi: 10.3389/fimmu.2022.988069. eCollection 2022.
Abstract
[This corrects the article DOI: 10.3389/fimmu.2022.803362.].
摘要
[本文更正了文章的数字对象标识符：10.3389/fimmu.2022.803362。]
相似文献
1
Corrigendum: Increased expression of LncRNA regulates the cigarette smoke and COPD associated airway inflammation and mucous cell hyperplasia.勘误：长链非编码RNA表达增加调节香烟烟雾和慢性阻塞性肺疾病相关的气道炎症及黏液细胞增生。
Front Immunol. 2022 Aug 8;13:988069. doi: 10.3389/fimmu.2022.988069. eCollection 2022.
2
Increased Expression of lncRNA Regulates the Cigarette Smoke and COPD Associated Airway Inflammation and Mucous Cell Hyperplasia.长链非编码 RNA 的表达增加调节香烟烟雾和 COPD 相关的气道炎症和黏液细胞增生。
Front Immunol. 2022 Jun 14;13:803362. doi: 10.3389/fimmu.2022.803362. eCollection 2022.
3
Expression of long non-coding RNA LUCAT1 in patients with chronic obstructive pulmonary disease and its potential functions in regulating cigarette smoke extract-induced 16HBE cell proliferation and apoptosis.长链非编码 RNA LUCAT1 在慢性阻塞性肺疾病患者中的表达及其在调节香烟烟雾提取物诱导的 16HBE 细胞增殖和凋亡中的潜在作用。
J Clin Lab Anal. 2021 Jul;35(7):e23823. doi: 10.1002/jcla.23823. Epub 2021 Jun 14.
4
Long non-coding RNA TUG1 promotes airway remodelling by suppressing the miR-145-5p/DUSP6 axis in cigarette smoke-induced COPD.长链非编码 RNA TUG1 通过抑制香烟烟雾诱导的 COPD 中的 miR-145-5p/DUSP6 轴促进气道重塑。
J Cell Mol Med. 2019 Nov;23(11):7200-7209. doi: 10.1111/jcmm.14389. Epub 2019 Sep 26.
5
Corrigendum: Autophagy Promotes Cigarette Smoke-Initiated and Elastin-Driven Bronchitis-Like Airway Inflammation in Mice.勘误：自噬促进香烟烟雾引发及弹性蛋白驱动的小鼠支气管炎样气道炎症。
Front Immunol. 2021 Sep 27;12:772939. doi: 10.3389/fimmu.2021.772939. eCollection 2021.
6
MTOR Suppresses Cigarette Smoke-Induced Epithelial Cell Death and Airway Inflammation in Chronic Obstructive Pulmonary Disease.雷帕霉素抑制香烟烟雾诱导的慢性阻塞性肺疾病上皮细胞死亡和气道炎症。
J Immunol. 2018 Apr 15;200(8):2571-2580. doi: 10.4049/jimmunol.1701681. Epub 2018 Mar 5.
7
Andrographolide antagonizes the cigarette smoke-induced epithelial-mesenchymal transition and pulmonary dysfunction through anti-inflammatory inhibiting HOTAIR.穿心莲内酯通过抗炎抑制 HOTAIR 拮抗香烟烟雾诱导的上皮-间充质转化和肺功能障碍。
Toxicology. 2019 Jun 15;422:84-94. doi: 10.1016/j.tox.2019.05.009. Epub 2019 May 23.
8
Epithelium-derived IL17A Promotes Cigarette Smoke-induced Inflammation and Mucus Hyperproduction.上皮细胞衍生的白介素 17A 促进香烟烟雾引起的炎症和黏液高分泌。
Am J Respir Cell Mol Biol. 2021 Dec;65(6):581-592. doi: 10.1165/rcmb.2020-0424OC.
9
Cigarette Smoke Activates NOTCH3 to Promote Goblet Cell Differentiation in Human Airway Epithelial Cells.香烟烟雾激活 NOTCH3 促进人呼吸道上皮细胞中的杯状细胞分化。
Am J Respir Cell Mol Biol. 2021 Apr;64(4):426-440. doi: 10.1165/rcmb.2020-0302OC.
10
Remodeling in response to infection and injury. Airway inflammation and hypersecretion of mucus in smoking subjects with chronic obstructive pulmonary disease.对感染和损伤的重塑。慢性阻塞性肺疾病吸烟患者的气道炎症和黏液分泌过多。
Am J Respir Crit Care Med. 2001 Nov 15;164(10 Pt 2):S76-80. doi: 10.1164/ajrccm.164.supplement_2.2106067.
Zotero 插件