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一种柳氮磺胺吡啶靶向前药可提高柳氮磺胺吡啶重新定位为抗结肠炎药物的治疗效果和安全性。

A Colon-Targeted Prodrug of Riluzole Improves Therapeutic Effectiveness and Safety upon Drug Repositioning of Riluzole to an Anti-Colitic Drug.

机构信息

College of Pharmacy, Pusan National University, Busan 46241, Republic of Korea.

出版信息

Mol Pharm. 2022 Nov 7;19(11):3784-3794. doi: 10.1021/acs.molpharmaceut.2c00255. Epub 2022 Aug 31.

Abstract

Riluzole (RLZ) is a neuroprotective drug indicated for amyotrophic lateral sclerosis. To examine the feasibility of RLZ for repositioning as an anti-inflammatory bowel disease (IBD) drug, RLZ (2, 5, and 10 mg/kg) was administered orally to rats with colitis induced by 2,4-dinitrobenzenesulfonic acid. Oral RLZ was effective against rat colitis in a dose-dependent manner, which was statistically significant at doses over 5 mg/kg. To address safety issues upon repositioning and further improve anti-colitic effectiveness, RLZ was coupled with salicylic acid (SA) via an azo-bond to yield RLZ-azo-SA (RAS) for the targeted colonic delivery of RLZ. Upon oral gavage, RAS (oral RAS) was efficiently delivered to and activated to RLZ in the large intestine, and systemic absorption of RLZ was substantially reduced. Oral RAS ameliorated colonic damage and inflammation in rat colitis and was more effective than oral RLZ and sulfasalazine, a current anti-IBD drug. Moreover, oral RAS potently inhibited glycogen synthase kinase 3β (GSK3β) in the inflamed distal colon, leading to the suppression of NFκB activity and an increase in the level of the anti-inflammatory cytokine interleukin-10. Taken together, RAS, which enables RLZ to be delivered to and inhibit GSK3β in the inflamed colon, may facilitate repositioning of RLZ as an anti-IBD drug.

摘要

利鲁唑(RLZ)是一种神经保护药物,用于治疗肌萎缩侧索硬化症。为了研究 RLZ 作为抗炎性肠病(IBD)药物重新定位的可行性,用 2,4-二硝基苯磺酸诱导大鼠结肠炎,以 RLZ(2、5 和 10mg/kg)进行口服给药。口服 RLZ 对大鼠结肠炎具有剂量依赖性疗效,在 5mg/kg 以上剂量时具有统计学意义。为了解重新定位的安全性问题并进一步提高抗结肠炎的疗效,将 RLZ 与水杨酸(SA)通过偶氮键连接,得到 RLZ-偶氮-SA(RAS),以实现 RLZ 在结肠的靶向递药。口服给予 RAS(口服 RAS)后,可有效递送至并在大肠中被激活为 RLZ,同时 RLZ 的全身吸收明显减少。口服 RAS 可改善大鼠结肠炎的结肠损伤和炎症,其疗效优于口服 RLZ 和柳氮磺胺吡啶,后者是一种目前的 IBD 药物。此外,口服 RAS 可有效抑制炎症性远端结肠中的糖原合成激酶 3β(GSK3β),从而抑制 NFκB 活性并增加抗炎细胞因子白细胞介素-10 的水平。总之,RAS 可使 RLZ 递送至炎症结肠并抑制 GSK3β,可能有助于 RLZ 作为 IBD 药物的重新定位。

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