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评价天然化合物木樨草素对人溶菌酶纤维形成的抑制潜力。

Evaluating the inhibitory potential of natural compound luteolin on human lysozyme fibrillation.

机构信息

Interdisciplinary Biotechnology Unit, Aligarh Muslim University Aligarh, UP, India.

Weill Cornell Medicine Qatar, Qatar Foundation, Education City, P.O. Box 24144, Doha, Qatar.

出版信息

Int J Biol Macromol. 2023 Apr 1;233:123623. doi: 10.1016/j.ijbiomac.2023.123623. Epub 2023 Feb 10.

Abstract

Numerous pathophysiological conditions known as amyloidosis, have been connected to protein misfolding leading to aggregation of proteins. Inhibition of cytotoxic aggregates or disaggregation of the preformed fibrils is thus one of the important strategies in the prevention of such diseases. Growing interest and exploration of identification of small molecules mainly natural compounds can prevent or delay amyloid fibril formation. We examined the mechanism of interaction and inhibition of human lysozyme (HL) aggregates with luteolin (LT). Biophysical and computational approaches have been employed to study the effect of LT on HL amyloid aggregation. Transmission Electronic Microscopy, Thioflavin T fluorescence, UV-vis spectroscopy, and RLS demonstrates that LT inhibit HL fibril formation. ANS fluorescence and hemolytic assay was also employed to examine the effect of the LT on toxicity of HL aggregation. Docking and molecular dynamics results showed that LT interacted with HL via hydrophobic and hydrogen interactions, thus reducing fibrillation levels. These findings highlight the benefit of polyphenols as safe therapy for preventing amyloid related diseases.

摘要

许多被称为淀粉样变性的病理生理状况都与蛋白质错误折叠导致蛋白质聚集有关。因此,抑制细胞毒性聚集体或解聚预先形成的纤维是预防此类疾病的重要策略之一。人们对鉴定小分子(主要是天然化合物)以预防或延迟淀粉样纤维形成的兴趣日益浓厚。我们研究了木樨草素(LT)与人溶菌酶(HL)聚集体相互作用和抑制的机制。采用生物物理和计算方法研究了 LT 对 HL 淀粉样聚集的影响。透射电子显微镜、硫黄素 T 荧光、紫外可见光谱和 RLS 表明 LT 抑制 HL 纤维形成。ANS 荧光和溶血试验也用于研究 LT 对 HL 聚集毒性的影响。对接和分子动力学结果表明,LT 通过疏水相互作用和氢键与 HL 相互作用,从而降低了纤维形成水平。这些发现强调了多酚作为预防与淀粉样相关疾病的安全疗法的益处。

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