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BRCA1/2 驱动的卵巢癌经新辅助化疗治疗后残留肿瘤的起源:预先存在的 BRCA1/2 功能肿瘤细胞的选择,而不是第二个 ORF 修复突变的获得。

Origin of Residual Tumor Masses in BRCA1/2-Driven Ovarian Carcinomas Treated by Neoadjuvant Chemotherapy: Selection of Preexisting BRCA1/2-Proficient Tumor Cells but Not the Gain of Second ORF-Restoring Mutation.

机构信息

Department of Tumor Growth Biology, N.N. Petrov Institute of Oncology, St. Petersburg, Russian Federation.

Department of Medical Genetics, St. Petersburg Pediatric Medical University, St. Petersburg, Russian Federation.

出版信息

Pathobiology. 2024;91(2):108-113. doi: 10.1159/000533591. Epub 2023 Aug 14.

Abstract

INTRODUCTION

Tubo-ovarian carcinomas (OCs) are highly sensitive to platinum-based neoadjuvant chemotherapy (NACT) but almost never demonstrate complete pathologic response.

METHODS

We analyzed paired primary and residual tumor tissues from 30 patients with hereditary BRCA1/2-driven OCs (BRCA1: 17; BRCA2: 13), who were treated by carboplatin/paclitaxel NACT (median number of cycles: 3, range: 3-6). BRCA1/2 and TP53 genes were analyzed by the next-generation sequencing. The ratio between TP53 mutation-specific versus wild-type reads was considered to monitor the proportion of tumor and non-tumor cells in the tissue sample, and the ratio between BRCA1/2-mutated and wild-type reads was used to estimate the presence of cells with the loss or retention of heterozygosity (LOH or ROH, respectively).

RESULTS

All 30 OCs had BRCA1/2 LOH in primary tumor and carried somatic TP53 mutation. Twenty-eight OCs had sufficient tumor cell cellularity in the post-NACT tissue to evaluate the ratio between mutated and wild-type BRCA1/2 alleles. Five (18%) out of 28 informative tumor pairs showed transition from LOH to ROH during NACT presumably affecting all or the vast majority of residual tumor cells. There were no signals of the emergence of a second open reading frame-restoring BRCA1/2 mutation.

CONCLUSION

Chemonaive BRCA1/2-driven carcinomas may contain a fraction of tumor cells with preserved BRCA1/2 heterozygosity. NACT can cause a selection of pre-existing BRCA1/2-proficient tumor cells, without gaining secondary reversal BRCA1/2 mutations.

摘要

简介

输卵管卵巢癌(OC)对铂类新辅助化疗(NACT)高度敏感,但几乎从未表现出完全的病理缓解。

方法

我们分析了 30 例遗传性 BRCA1/2 驱动的 OC 患者(BRCA1:17 例;BRCA2:13 例)的配对原发和残留肿瘤组织,这些患者接受了卡铂/紫杉醇 NACT(中位周期数:3 个,范围:3-6 个)治疗。通过下一代测序分析 BRCA1/2 和 TP53 基因。突变特异性与野生型读码的比例被认为可以监测组织样本中肿瘤细胞和非肿瘤细胞的比例,而 BRCA1/2 突变型与野生型读码的比例则用于估计存在缺失或保留杂合性(分别为 LOH 或 ROH)的细胞比例。

结果

所有 30 例 OC 在原发肿瘤中均存在 BRCA1/2 LOH,并携带体细胞 TP53 突变。28 例 OC 在 NACT 后组织中有足够的肿瘤细胞细胞密度来评估突变和野生型 BRCA1/2 等位基因之间的比例。28 对有意义的肿瘤对中有 5 对(18%)在 NACT 期间从 LOH 转变为 ROH,推测这可能影响所有或绝大多数残留肿瘤细胞。没有出现第二个开放阅读框恢复 BRCA1/2 突变的信号。

结论

化疗前的 BRCA1/2 驱动的癌可能包含一部分保留 BRCA1/2 杂合性的肿瘤细胞。NACT 可以选择预先存在的 BRCA1/2 功能良好的肿瘤细胞,而不会获得继发的 BRCA1/2 逆转突变。

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