• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

[心理社会风险对幼儿脑损伤儿童脑电图背景活动成熟的影响]

[Effect of psychosocial risks on the maturation of EEG background activity in early childhood brain-damaged children].

作者信息

Cammann R, Göllnitz G, Cammann G, Heider B, Meyer-Probst B, Teichmann H

出版信息

Z Kinder Jugendpsychiatr. 1985;13(1):5-15.

PMID:3837560
Abstract

Age-dependent changes in EEG activity were assessed in a comparison of EEG recordings taken from 102 children at the ages of 6 and 10 years. Of the 2-to-13-cycle-a-second activity recorded from the left occipital region, the fast alpha frequencies (10 to 13 cycles a second) showed a significant increase at the expense of all slower frequencies. The children were divided into two groups according to whether they had been exposed to a small or large number of biological risks; in the latter group the extent of the increase in fast alpha activity was negatively related to the number of additional psychosocial risks. This effect was even more pronounced in the group of brain-damaged children, where the smallest age-dependent shift in the EEG frequencies was seen in the group with both minimal brain damage and a high psychosocial risk factor. These data suggest that the exposure of children with minimal brain damage to multiple psychosocial risks may lead to a delay in the maturation of the brain that can be detected by neurophysiological methods.

摘要

通过比较102名6岁和10岁儿童的脑电图记录,评估了脑电图活动的年龄依赖性变化。在从左枕叶区域记录的每秒2至13次循环的活动中,快速阿尔法频率(每秒10至13次循环)显著增加,而所有较慢频率的活动则相应减少。根据儿童所面临的生物风险数量的多少,将他们分为两组;在生物风险数量较多的那组儿童中,快速阿尔法活动的增加程度与额外心理社会风险的数量呈负相关。这种效应在脑损伤儿童组中更为明显,在脑损伤最小且心理社会风险因素较高的组中,脑电图频率随年龄的变化最小。这些数据表明,轻度脑损伤儿童暴露于多种心理社会风险中,可能会导致大脑成熟延迟,这种延迟可以通过神经生理学方法检测到。

相似文献

1
[Effect of psychosocial risks on the maturation of EEG background activity in early childhood brain-damaged children].[心理社会风险对幼儿脑损伤儿童脑电图背景活动成熟的影响]
Z Kinder Jugendpsychiatr. 1985;13(1):5-15.
2
Increased event-related theta activity as a psychophysiological marker of comorbidity in children with tics and attention-deficit/hyperactivity disorders.事件相关θ活动增加作为抽动秽语综合征和注意缺陷多动障碍共病儿童的心理生理标志物
Neuroimage. 2006 Aug 15;32(2):940-55. doi: 10.1016/j.neuroimage.2006.03.056. Epub 2006 May 26.
3
Coherence in children with Attention-Deficit/Hyperactivity Disorder and excess beta activity in their EEG.注意缺陷多动障碍儿童的脑电相干性及脑电图中β活动过多
Clin Neurophysiol. 2007 Jul;118(7):1472-9. doi: 10.1016/j.clinph.2007.04.006. Epub 2007 May 14.
4
EEG coherence in children with attention-deficit/hyperactivity disorder and comorbid reading disabilities.注意缺陷多动障碍合并阅读障碍儿童的脑电图相干性
Int J Psychophysiol. 2009 Mar;71(3):205-10. doi: 10.1016/j.ijpsycho.2008.09.003. Epub 2008 Sep 25.
5
The visual scoring of sleep and arousal in infants and children.婴幼儿睡眠与觉醒的视觉评分
J Clin Sleep Med. 2007 Mar 15;3(2):201-40.
6
Hyperkinetic disorder in the ICD-10: EEG evidence for a definitional widening?
Eur Child Adolesc Psychiatry. 2003 Apr;12(2):92-9. doi: 10.1007/s00787-003-0315-5.
7
A study on the late consequences of a brain trauma in early childhood (BTEC).
Psychiatr Neurol Med Psychol Beih. 1968;8-9:60-3.
8
Interacting effects of the dopamine transporter gene and psychosocial adversity on attention-deficit/hyperactivity disorder symptoms among 15-year-olds from a high-risk community sample.来自高危社区样本的15岁青少年中,多巴胺转运体基因与心理社会逆境对注意力缺陷多动障碍症状的交互作用。
Arch Gen Psychiatry. 2007 May;64(5):585-90. doi: 10.1001/archpsyc.64.5.585.
9
[Development of bioelectric activity in children 7 to 11 years old].[7至11岁儿童生物电活动的发展]
Psychiatr Neurol Med Psychol Beih. 1970;13-14:223-8.
10
Resting EEG theta activity predicts cognitive performance in attention-deficit hyperactivity disorder.静息脑电图θ波活动可预测注意缺陷多动障碍的认知表现。
Pediatr Neurol. 2005 Apr;32(4):248-56. doi: 10.1016/j.pediatrneurol.2004.11.009.