Riabinin V E, Lifshits R I
Vopr Med Khim. 1985 Sep-Oct;31(5):38-40.
Induction of NADPH- and ascorbate-dependent lipid peroxidation has been shown to cause a decrease in the rate of oxygen utilization by liver microsomes of burned mice as compared with controls. The thermal injury did not change the activity of NADPH- and NADH-oxidoreductases, thus indicating that initial elements of electron transfer system were still maintained at the normal state.
与对照组相比，已证明诱导依赖烟酰胺腺嘌呤二核苷酸磷酸（NADPH）和抗坏血酸的脂质过氧化会导致烧伤小鼠肝脏微粒体的氧利用率降低。热损伤并未改变NADPH和烟酰胺腺嘌呤二核苷酸（NADH）氧化还原酶的活性，因此表明电子传递系统的初始元件仍维持在正常状态。
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