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1
An attempt at an analysis of the factors determining the time course of the end-plate current. I. The effects of prostigmine and of the ratio of Mg 2+ to Ca 2+ .
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2
An attempt at an analysis of the factors determining the time course of the end-plate current. II. Temperature.
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3
The effect of voltage on the time course of end-plate currents.
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4
A comparison of the effect of cholinesterase inhibitors on end-plate current and on cholinesterase activity in frog muscle.
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Effect of prostigmine on the time course of the end-plate potential in the rat diaphragm.
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6
On the role of junctional cholinesterase in determining the time course of the end-plate current.
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Effects of muscle stretch on transmitter release at end-plates of rat diaphragm and frog sartorius muscle.
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8
Effects of 4-aminopyridine at the frog neuromuscular junction.
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9
Effect of repetitive stimulation on the frog neuromuscular transmission.
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Analysis of the effects of histamine on the end-plate potential.
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1
Mechanisms shaping the slow nicotinic synaptic current at the motoneuron-renshaw cell synapse.
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2
The kinetics of local anesthetic blockade of end-plate channels.
Biophys J. 1982 Mar;37(3):625-31.
3
Voltage clamp study of fast excitatory synaptic currents in bullfrog sympathetic ganglion cells.
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The voltage and temperature dependence of the end-plate current in frog skeletal muscle.
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End-plate currents evoked by paired stimuli in frog muscle fibres.
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Inhibition of acetylcholine responses by intracellular calcium in Lymnaea stagnalis neurones.
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A covalently bound photoisomerizable agonist: comparison with reversibly bound agonists at Electrophorus electroplaques.
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An analysis of the relationship between the current and potential generated by a quantum of acetylcholine in muscle fibers without transverse tubules.
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The effect of anticholinesterase drugs on the ionophoretically-evoked end-plate currents.
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本文引用的文献
1
Actions of anti-cholinesterases on endplate potential of frog muscle.
J Neurophysiol. 1949 Jan;12(1):59-80. doi: 10.1152/jn.1949.12.1.59.
2
Interaction between sodium and calcium ions in the process of transmitter release at the neuromuscular junction.
J Physiol. 1968 Sep;198(1):203-18. doi: 10.1113/jphysiol.1968.sp008602.
3
An analysis of the end-plate potential recorded with an intracellular electrode.
J Physiol. 1951 Nov 28;115(3):320-70. doi: 10.1113/jphysiol.1951.sp004675.
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Hyperpolarization of mammalian motor nerve terminals.
J Physiol. 1962 Aug;163(1):115-37. doi: 10.1113/jphysiol.1962.sp006961.
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THE EFFECT OF CALCIUM ON ACETYLCHOLINE RELEASE FROM MOTOR NERVE TERMINALS.
Proc R Soc Lond B Biol Sci. 1965 Feb 16;161:496-503. doi: 10.1098/rspb.1965.0017.
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Active phase of frog's end-plate potential.
J Neurophysiol. 1959 Jul;22(4):395-411. doi: 10.1152/jn.1959.22.4.395.
7
The nature of the antagonism between calcium and magnesium ions at the neuromuscular junction.
J Physiol. 1957 Oct 30;138(3):434-44. doi: 10.1113/jphysiol.1957.sp005860.
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Statistical factors involved in neuromuscular facilitation and depression.
J Physiol. 1954 Jun 28;124(3):574-85. doi: 10.1113/jphysiol.1954.sp005130.
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Post-tetanic restoration of neuromuscular transmission blocked by D-tubocurarine.
J Physiol. 1952 Oct;118(2):216-27. doi: 10.1113/jphysiol.1952.sp004788.
10
Analysis of sodium and potassium conductances in the procaine end-plate potential.
J Physiol. 1966 Apr;183(3):592-606. doi: 10.1113/jphysiol.1966.sp007886.
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