Steriade M, Kitsikis A, Oakson G
Sleep. 1979 Summer;1(4):339-55. doi: 10.1093/sleep/1.4.339.
Excitatory-inhibitory processes of parietal association neurons were studied during reticular-induced EEG activation and natural sleep-waking cycle. The probability of antidromic invasion in long-axon cells is enhanced following midbrain reticular stimulation and during both waking (W) and desynchronized sleep (D) states compared to slow-wave sleep. The thalamically elicited inhibitory phase occurs with a shorter latency following reticular stimulation and during W and D states; this is due to the reduced duration, during all these experimental conditions, of secondary excitatory processes. The duration of the inhibitory period is shorter during reticular activation; the postinhibitory rebound occurs at shorter latencies and is sharper following reticular stimulation and during W and D states. The similarity between reticular effects and changes during both W and D states is discussed.
在网状结构诱导的脑电图激活和自然睡眠-觉醒周期过程中，对顶叶联合神经元的兴奋-抑制过程进行了研究。与慢波睡眠相比，在中脑网状结构刺激后以及清醒（W）和去同步化睡眠（D）状态下，长轴突细胞中逆向入侵的概率会增加。在网状结构刺激后以及W和D状态下，丘脑诱发的抑制期潜伏期较短；这是由于在所有这些实验条件下，继发性兴奋过程的持续时间缩短。在网状结构激活期间，抑制期的持续时间较短；抑制后反弹在较短潜伏期出现，并且在网状结构刺激后以及W和D状态下更为明显。文中讨论了网状结构效应与W和D状态下变化之间的相似性。
