Kertesz A
Cortex. 1979 Sep;15(3):403-19. doi: 10.1016/s0010-9452(79)80067-2.
This case of visual agnosia is of special interest because of its causation by trauma, of the unusually long follow-up (10 1/2 years), and the evidence for dual deficits of recognition and perception. Although most of the findings were characteristic of associative visual agnosia with preserved perceptual function, the poor copying, contrasted to better spontaneous drawing, suggested apperceptive agnosia as well. Prosopagnosia, alexia without agraphia, Balint's syndrome, visual static agnosia and simultanagnosia were also observed. The patient had persisting amnestic syndrome, but no dementia or aphasia. The responses to visual stimulation were perseverations, form confusions and confabulations. Visual evoked potentials were severely, bilaterally abnormal and computerized tomographic localization showed bilateral lesions also. The stages of recognition are analysed through this case of visual verbal disconnection and the importance of memory in perception is highlighted.
该例视觉失认症因其由创伤引起、随访时间异常长（10年半）以及存在识别和感知双重缺陷的证据而具有特殊意义。尽管大多数发现具有知觉功能保留的联想性视觉失认症的特征，但与较好的自发绘图相比，临摹能力较差提示也存在apperceptive失认症。还观察到面孔失认症、单纯性失读症、Balint综合征、视觉静态失认症和同时失认症。患者存在持续性遗忘综合征，但无痴呆或失语。对视觉刺激的反应为持续反应、形状混淆和虚构。视觉诱发电位严重双侧异常，计算机断层扫描定位也显示双侧病变。通过该例视觉言语分离病例分析了识别阶段，并强调了记忆在感知中的重要性。
