Stöhr M, Buettner U W, Riffel B, Koletzki E
Electroencephalogr Clin Neurophysiol. 1982 Sep;54(3):257-65. doi: 10.1016/0013-4694(82)90175-4.
Short-latency somatosensory evoked potentials (SEPs) to median and ulnar nerve stimulation were studied in 17 patients with cervical cord lesions of different sites and origins. Spinal cord lesions involving the 6th and 7th cervical segment led to an attenuation or loss of the cervical response after stimulation of the median nerve, whereas lesions at C8/D1 only affected the cervical response evoked by ulnar nerve stimulation. The third component of the neck-SEP (N13) was exclusively involved in some cases. More extensive lesions additionally abolished the second component (N11). Spinal cord lesions above the 6th cervical segment caused some dissociation of the simultaneously recorded responses at C7 and C2, with preserved responses at C7 and disturbed or absent evoked potentials at C2.
对17例不同部位和病因的颈髓损伤患者进行了正中神经和尺神经刺激下的短潜伏期体感诱发电位(SEP)研究。累及颈6和颈7节段的脊髓损伤导致正中神经刺激后颈段反应减弱或消失,而C8/D1节段的损伤仅影响尺神经刺激诱发的颈段反应。在某些病例中,颈SEP的第三个成分(N13)单独受累。更广泛的损伤还会使第二个成分(N11)消失。颈6节段以上的脊髓损伤导致同时记录的C7和C2反应出现一些分离,C7反应保留,而C2的诱发电位受到干扰或消失。