Aliot E, Saulnier J P, Bruntz J F, Chevrier J, Gregoire P, Gilgenkrantz J M
Arch Mal Coeur Vaiss. 1982 May;75(5):513-9.
Bidirectional ventricular tachycardia, defined as the rapid alternation of the QRS complexes with successive opposing axial deviation, is a rare arrhythmia. In the rare cases which have undergone endocavitary investigations, an infrahisian origin has generally been proved. However, the mechanism of these tachycardias remains poorly understood and is discussed with respect to a new case. Bidirectional tachycardia occurred in a 79 year old woman with previous diaphragmatic and anterior wall infarction. It was a wide QRS tachycardia at 180/min with a succession of ventriculogrammes of opposing axis in the frontal plane and permanent right bundle branch block over the right precordium. The two types of tachycardia were observed, monomorphic type A or Type B or a combination of the two realising an A-B bidirectional tachycardia. The origin of these episodes, which occurred on a background of atrial tachycardia at about 100/min, was ventricular as shown by the absence of a His potential before the ventricular complexes in tachycardia. The presence of ventricular extrasystoles with relatively fixed coupling intervals, and the results of endocavitary investigation were suggestive of a reentry phenomenon ventricular extrastimuli were capable of transforming the bidirectional into monomorphic tachycardia and vice versa; this suggests that A was at times the origin of a reentry B, but protected by A, tachycardia B could be sustained. In the light of previously reported cases with documented endocavitary investigation and this new case, it seems possible to talk in terms of true "bidirectional ventricular tachycardia", a tachycardia whose mechanism is obscure but certainly not univocal.
双向性室性心动过速定义为QRS波群快速交替且相继出现相反的电轴偏移,是一种罕见的心律失常。在少数接受心腔内检查的病例中,通常已证实其起源于希氏束以下。然而,这些心动过速的机制仍知之甚少,本文结合一个新病例进行讨论。一名79岁曾有膈面及前壁梗死的女性发生了双向性心动过速。这是一种宽QRS波心动过速,频率为180次/分钟,额面导联上心电轴相继出现相反方向的变化,右胸前导联持续存在右束支传导阻滞。观察到两种类型的心动过速,即单形性A型或B型,或两者结合形成A - B型双向性心动过速。这些发作起源于心房心动过速(频率约为100次/分钟)背景下的心室,心动过速时心室复合波前无希氏束电位即可证明。存在配对间期相对固定的室性期前收缩,心腔内检查结果提示存在折返现象,室性期外刺激能够将双向性心动过速转变为单形性心动过速,反之亦然;这表明有时A是折返B的起源,但受A的保护,心动过速B得以持续。根据先前报道的有记录的心腔内检查病例以及这个新病例,似乎可以提及真正的“双向性室性心动过速”,这种心动过速的机制尚不清楚,但肯定不是单一的。