钾通道调节大鼠神经末梢神经瘤的异位兴奋性。
Potassium channels moderate ectopic excitability of nerve-end neuromas in rats.
作者信息
Devor M
出版信息
Neurosci Lett. 1983 Sep 30;40(2):181-6. doi: 10.1016/0304-3940(83)90299-9.
PMID:6314210
Abstract
The potassium conductance (gK) blockers tetraethylammonium (TEA) and 4-aminopyridine (4-AP) strongly excited experimental neuromas that had spontaneous discharge, but had minimal effects on acutely cut nerves and on old, quiescent neuromas. Resting gK apparently moderates hyperexcitability in active neuromas.
摘要
钾离子电导（gK）阻滞剂四乙铵（TEA）和4-氨基吡啶（4-AP）强烈兴奋具有自发放电的实验性神经瘤，但对急性切断的神经和陈旧、静止的神经瘤影响极小。静息gK显然可调节活跃神经瘤的过度兴奋性。
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