缺血性缺氧所致脑损伤：假说扩展——两种离子的故事？
Brain injury by ischemic anoxia: hypothesis extension--a tale of two ions?
作者信息
White B C, Aust S D, Arfors K E, Aronson L D
出版信息
Ann Emerg Med. 1984 Sep;13(9 Pt 2):862-7. doi: 10.1016/s0196-0644(84)80461-8.
PMID:6476556
Abstract
The understanding of the pathophysiology of cerebral ischemic anoxia depends on elucidation of cellular neuronal changes during this event and under conditions of cardiopulmonary resuscitation. Current knowledge of these metabolic developments is reviewed, and a hypothesis of the role of iron in this situation is presented.
摘要
对脑缺血缺氧病理生理学的理解取决于对该事件期间以及心肺复苏条件下细胞神经元变化的阐明。本文综述了目前关于这些代谢进展的知识，并提出了铁在这种情况下作用的假说。
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