Resende L A, Battistuta R J, Sato T, De Bortoli N A
Arq Neuropsiquiatr. 1984 Jun;42(2):171-4. doi: 10.1590/s0004-282x1984000200011.
The authors report a case of a 27 year old male patient who has exhibited external ophthalmoplegia, anisocoria, ataxia, absent deep reflexes, dysphagia, dysphonia, bilateral peripheral facial paralysis, hypopallesthesia and a mild sensory impairment of the four extremities. In addition, his cerebrospinal fluid showed albuminocytologic dissociation. The otoneurologic examination showed important findings. These symptoms began ten days after a "cold", and quickly subsided after prednisone therapy. Two months after the onset of clinical features, just a deep apendicular areflexia was detected on his neurological examination. The neurological signs and complementary laboratory findings suggest that both central and peripheral alterations are responsible for the clinical manifestations.
作者报告了一例27岁男性患者,该患者出现了眼球外肌麻痹、瞳孔不等大、共济失调、深部反射消失、吞咽困难、发音障碍、双侧周围性面瘫、轻触觉减退以及四肢轻度感觉障碍。此外,他的脑脊液显示蛋白细胞分离。耳神经学检查有重要发现。这些症状在“感冒”后十天开始出现,泼尼松治疗后迅速消退。临床症状出现两个月后,其神经学检查仅发现深部肢体反射消失。神经体征和辅助实验室检查结果表明,中枢和外周改变均导致了临床表现。