在枯草芽孢杆菌的混合感染中，噬菌体SP82比噬菌体SP01占优势。
Predominance of bacteriophage SP82 over bacteriophage SP01 in mixed infections of Bacillus subtilis.
作者信息
Stewart C R, Franck M
出版信息
J Virol. 1981 Jun;38(3):1081-3. doi: 10.1128/JVI.38.3.1081-1083.1981.
Abstract
In mixed infections with Bacillus subtilis phages SP82 and SP01, the SP82 genotype is predominant among the progeny. This predominance is determined by a specific region of the genome, the pos region, which apparently is located near genes 29 to 32 (by the SP01 numbering system). Recombination between SP82 and SP01 yields phage which have both the SP82 pos region and an SP01 mutation. This mutation then behaves in mixed infection as if it were part of an SP82 genome.
摘要
在枯草芽孢杆菌噬菌体SP82和SP01的混合感染中，SP82基因型在子代中占主导地位。这种优势由基因组的一个特定区域——pos区域决定，该区域显然位于基因29至32附近（按照SP01编号系统）。SP82和SP01之间的重组产生的噬菌体同时具有SP82的pos区域和一个SP01突变。在混合感染中，这个突变的表现就好像它是SP82基因组的一部分。
相似文献
1
Predominance of bacteriophage SP82 over bacteriophage SP01 in mixed infections of Bacillus subtilis.
J Virol. 1981 Jun;38(3):1081-3. doi: 10.1128/JVI.38.3.1081-1083.1981.
2
Bacillus subtilis bacteriophage SP01, SP82, and phi e require host lysyl- and tryptophanyl-tRNA synthetases for phage development.
J Virol. 1974 Aug;14(2):402-6. doi: 10.1128/JVI.14.2.402-406.1974.
3
Mixed infections of Bacillus subtilis involving bacteriophages SP82 and beta 22.
J Virol. 1974 Dec;14(6):1463-9. doi: 10.1128/JVI.14.6.1463-1469.1974.
4
Comparison of the physical maps and redundant ends of the chromosomes of phages 2C, SP01, SP82 and phi e.
Eur J Biochem. 1983 Apr 15;132(1):63-7. doi: 10.1111/j.1432-1033.1983.tb07325.x.
5
Recombination and expression of a cloned fragment of the DNA of Bacillus subtilis bacteriophage SP01.
Virology. 1982 Jul 30;120(2):307-17. doi: 10.1016/0042-6822(82)90032-0.
6
Dominance relationships in mixedly infected Bacillus subtilis.
J Virol. 1972 Apr;9(4):594-601. doi: 10.1128/JVI.9.4.594-601.1972.
7
Early RNAs in SP82- and SP01-infected Bacillus subtilis may be processed.
J Virol. 1981 Mar;37(3):1075-8. doi: 10.1128/JVI.37.3.1075-1078.1981.
8
Mixed infections of Bacillus subtilis involving bacteriophage SPO2c 1 .
J Virol. 1973 Jan;11(1):25-34. doi: 10.1128/JVI.11.1.25-34.1973.
9
DNA gyrase inhibitors block development of Bacillus subtilis bacteriophage SP01.
J Virol. 1981 Sep;39(3):855-60. doi: 10.1128/JVI.39.3.855-860.1981.
10
Promoter recognition by phage SP01-modified RNA polymerase.
Proc Natl Acad Sci U S A. 1978 Mar;75(3):1185-9. doi: 10.1073/pnas.75.3.1185.
引用本文的文献
1
Group I intron homing in Bacillus phages SPO1 and SP82: a gene conversion event initiated by a nicking homing endonuclease.
J Bacteriol. 2004 Jul;186(13):4307-14. doi: 10.1128/JB.186.13.4307-4314.2004.
本文引用的文献
1
Bacteriophage T4 mutants unable to exclude gene 56 of T2 from the progeny of crosses.
Mutat Res. 1970 Mar;9(3):345-8. doi: 10.1016/0027-5107(70)90136-3.
2
RNA synthesis during bacteriophage SPO1 development: six classes of SPO1 RNA.
J Mol Biol. 1971 Apr 28;57(2):279-300. doi: 10.1016/0022-2836(71)90346-9.
3
[Comparative study of the DNA of phages 2C, SP8*, SP82, phi e, SP01 and SP50].
Eur J Biochem. 1970 Aug;15(2):391-400. doi: 10.1111/j.1432-1033.1970.tb01020.x.
4
Partial exclusion of genes of bacteriophage T2 with T4-glucosylated DNA in crosses with bacteriophage T4.
Genetica. 1970;41(4):541-50. doi: 10.1007/BF00958933.
5
Mixed infections of Bacillus subtilis involving bacteriophage SPO2c 1 .
J Virol. 1973 Jan;11(1):25-34. doi: 10.1128/JVI.11.1.25-34.1973.
6
DNA replication and late protein synthesis during SP82 infection of Bacillus subtilis.
Virology. 1972 Dec;50(3):653-63. doi: 10.1016/0042-6822(72)90419-9.
7
The genetics of bacteriophage SPO1.
Biken J. 1972 Jun;15(2):81-97.
8
Replication of viral DNA in SPO1-infected Bacillus subtilis. I. Replicative intermediates.
Virology. 1972 May;48(2):402-16. doi: 10.1016/0042-6822(72)90051-7.
9
Dominance relationships in mixedly infected Bacillus subtilis.
J Virol. 1972 Apr;9(4):594-601. doi: 10.1128/JVI.9.4.594-601.1972.
10
Partial exclusion between T-even bacteriophages: an incipient genetic isolation mechanism.
Genetics. 1974 Dec;78(4):989-1014. doi: 10.1093/genetics/78.4.989.
Zotero 插件