抗惊厥药所致佝偻病伴肾小管性酸中毒及血清1,25-二羟维生素D水平正常 - Suppr | 超能文献

Anticonvulsant-induced rickets associated with renal tubular acidosis and normal level of serum 1,25-dihydroxyvitamin D.

作者信息

Ishitsu T, Matsuda I, Seino Y, Kann H

出版信息

Am J Dis Child. 1981 Dec;135(12):1140-2. doi: 10.1001/archpedi.1981.02130360044016.

DOI:10.1001/archpedi.1981.02130360044016

Abstract

摘要

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Anticonvulsant-induced rickets associated with renal tubular acidosis and normal level of serum 1,25-dihydroxyvitamin D.抗惊厥药所致佝偻病伴肾小管性酸中毒及血清1,25-二羟维生素D水平正常

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Renal tubular acidosis and skeletal demineralization in patients on long-term anticonvulsant therapy.长期接受抗惊厥治疗患者的肾小管酸中毒和骨骼脱矿质作用。

J Pediatr. 1975 Aug;87(2):202-5. doi: 10.1016/s0022-3476(75)80579-8.

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Rickets precipitated by anticonvulsant drugs in a child receiving prophylactic vitamin D.在接受预防性维生素D治疗的儿童中，抗惊厥药物引发的佝偻病。

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Rickets following anticonvulsant therapy.抗惊厥治疗后发生的佝偻病

Proc R Soc Med. 1972 Aug;65(8):730. doi: 10.1177/003591577206500832.

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Rickets associated with anticonvulsant medication.与抗惊厥药物相关的佝偻病

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Long-term administration of antiepileptic drugs and the development of rickets.抗癫痫药物的长期使用与佝偻病的发生

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[Severe osteomalacic rickets induced by antiepileptic drugs. Literature review apropos of a case].[抗癫痫药物所致严重骨软化性佝偻病。结合一例病例的文献综述]

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[Distinct rickets during anticonvulsant long-term therapy].[抗惊厥长期治疗期间的独特佝偻病]

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Renal tubular acidosis: pathogenesis, diagnosis and treatment.肾小管酸中毒：发病机制、诊断与治疗

Indian J Pediatr. 1988 May-Jun;55(3):379-94. doi: 10.1007/BF02810360.

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Decreased bone density in severely handicapped children and adults, with reference to the influence of limited mobility and anticonvulsant medication.

Eur J Pediatr. 1986 Feb;144(5):457-63. doi: 10.1007/BF00441738.