Woodfield D G, Douglas R, Smith J, Simpson A, Pinder L, Staveley J M
Transfusion. 1982 Jul-Aug;22(4):276-8. doi: 10.1046/j.1537-2995.1982.22482251206.x.
The Kidd locus phenotype Jk(a-b-) was detected in 0.9 percent of Polynesians living in New Zealand. Over a period of 13 years, nine examples of anti-Jk3 were detected, one of which caused a delayed hemolytic transfusion reaction. Other examples resulted in mild hemolytic disease of the newborn. The anti-Jk3 reacted as an inseparable antibody, confirmed that inheritance of the Jk(a-b-) phenotype was best explained by the presence of a silent Jk allele.
在生活在新西兰的波利尼西亚人中,0.9%的人检测出基德血型系统Jk(a-b-)表型。在13年的时间里,检测到9例抗Jk3抗体,其中1例引发了迟发性溶血性输血反应。其他病例导致了新生儿轻度溶血病。抗Jk3抗体表现为不可分离的抗体,这证实Jk(a-b-)表型的遗传现象最能通过一个沉默的Jk等位基因的存在来解释。