Weinstein R S, Hudson J B
Arch Intern Med. 1980 Mar;140(3):410-1.
A 38-year-old man developed symptomatic hypercalcemia during the diuretic phase of acute renal failure secondary to rhabdomyolysis. Although early secondary hyperparathyroidism was documented, parathyroid hormone levels fell when hypercalcemia occurred. The 25-hydroxycholecalciferol level was normal on two separate occasions during the hypercalcemia. Remobilization of muscle calcium deposits during diuresis is proposed as the cause of the hypercalcemia in this syndrome.
一名38岁男性在继发于横纹肌溶解的急性肾衰竭利尿期出现症状性高钙血症。尽管记录到早期继发性甲状旁腺功能亢进，但高钙血症发生时甲状旁腺激素水平下降。在高钙血症期间，两次独立检测时25-羟胆钙化醇水平均正常。利尿期间肌肉钙沉积的再动员被认为是该综合征高钙血症的原因。
