直链烷基苯磺酸盐对斑节对虾的毒性作用。

Toxic effects of linear alkylbenzene sulfonate on the tiger prawn, Penaeus monodon.

作者信息

Hwang D F, Chen M Y, Yoshida T, Jeng S S

机构信息

Graduate School of Marine Food Science, National Taiwan Ocean University, Keelung, Republic of China.

出版信息

Ecotoxicol Environ Saf. 1993 Dec;26(3):285-92. doi: 10.1006/eesa.1993.1057.

DOI:10.1006/eesa.1993.1057

PMID:7507819

Abstract

The toxic effects of linear alkybenzene sulfonate (LAS) on the larvae and juveniles of tiger prawn Penaeus monodon were tested. The 24-hr LC50 values of LAS were 0.06, 0.10, and 3.11 ppm for the zoea 2nd substage, mysis 2nd substage (M2), and postlarva 12th substage (PL12) of the tiger prawn, respectively. The 48-hr LC50 values of LAS were 0.07, 1.03, and 4.36 ppm for M2, PL12, and postlarva 15th substage of the tiger prawn, respectively. The hepatopancreatic glutathione (GSH) in juvenile tiger prawn was obviously depleted when LAS concentration was over 1.0 ppm. The hepatopancreatic GSH content of tiger prawn exposed to solutions of greater than 1.0 ppm LAS was difficult to raise to normal levels, even after the tiger prawns were switched to an LAS-free solution. The activity of malate dehydrogenase in the serum of juvenile tiger prawns exposed to 10.0 ppm LAS solution was significantly increased.

摘要

测试了直链烷基苯磺酸盐（LAS）对斑节对虾幼体和稚虾的毒性作用。LAS对斑节对虾第二期蚤状幼体、第二期糠虾幼体（M2）和第十二期后期幼体（PL12）的24小时半数致死浓度（LC50）值分别为0.06、0.10和3.11 ppm。LAS对斑节对虾M2、PL12和第十五期后期幼体的48小时LC50值分别为0.07、1.03和4.36 ppm。当LAS浓度超过1.0 ppm时，斑节对虾稚虾的肝胰腺谷胱甘肽（GSH）明显消耗。即使将斑节对虾转移到不含LAS的溶液中，暴露于浓度大于1.0 ppm LAS溶液中的斑节对虾肝胰腺GSH含量也难以恢复到正常水平。暴露于10.0 ppm LAS溶液中的斑节对虾稚虾血清中苹果酸脱氢酶的活性显著增加。