Lampl Y, Gilad R, Eshel Y, Sarova-Pinhas I
Department of Neurology, Edith Wolfson Medical Center, Holon, Israel.
Clin Neurol Neurosurg. 1995 Aug;97(3):203-7. doi: 10.1016/0303-8467(95)00037-k.
Sensory-motor deficit in a peripheral nerve pattern due to brain lesion is rarely documented. We report on seven patients with a clinical manifestation of sensory-motor deficit, imitating peripheral nerve involvement, due to lacunar brain infarcts verified by brain computed tomography scan. Five of the patients had an ulnar nerve-like deficit and two median nerve-like deficits. The infarcts were located in the thalamus and the corona radiata. No clinical or electrophysiological evidence for peripheral nerve involvement was found. The unusual peripheral nerve pattern of lesions caused by lacunar brain infarcts can be defined as an additional lacunar syndrome and must be taken into consideration in the clinical evaluation of peripheral nerve deficits with normal nerve conduction velocity.
因脑损害导致的周围神经型感觉运动功能缺损鲜有文献记载。我们报告了7例临床表现为感觉运动功能缺损、酷似周围神经受累的患者,其病因经脑部计算机断层扫描证实为腔隙性脑梗死。其中5例患者有尺神经样缺损,2例有正中神经样缺损。梗死灶位于丘脑和放射冠。未发现周围神经受累的临床或电生理证据。腔隙性脑梗死所致的不寻常周围神经型损害可定义为一种额外的腔隙综合征,在临床评估神经传导速度正常的周围神经缺损时必须予以考虑。