一种对尾部表型具有短尾相关基因非依赖效应的新型部分t单倍型。
A novel partial t haplotype with a brachyury-independent effect on tail phenotype.
作者信息
Fujimoto A, Wakasugi N, Tomita T
机构信息
Laboratory of Animal Genetics, School of Agricultural Sciences, Nagoya University, Japan.
出版信息
Mamm Genome. 1995 Jun;6(6):396-400. doi: 10.1007/BF00355639.
The btm (brachyury-interacting tail length modifier) mutation was discovered in strain MOL-NIS derived from Japanese wild mice (Mus musculus molossinus) as an autosomal recessive mutation. Homozygotes for this mutation show a short tail phenotype and, moreover, this mutation causes the tailless character by interacting with the T (brachyury) gene on Chromosome (Chr) 17. Our linkage tests and RFLP analyses suggest that btm is located within the t complex on Chr 17 and represents a new partial t haplotype.
btm（短尾交互尾长修饰基因）突变是在源自日本野生小鼠（小家鼠）的MOL-NIS品系中作为常染色体隐性突变被发现的。该突变的纯合子表现出短尾表型，此外，此突变通过与17号染色体（Chr）上的T（短尾）基因相互作用导致无尾性状。我们的连锁试验和RFLP分析表明，btm位于17号染色体的t复合体内，代表一种新的部分t单倍型。
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