Induction of mixed bone marrow chimerism as potential therapy for autoimmune (type I) diabetes: experience in the NOD model.
作者信息
Mathieu C, Bouillon R, Rutgeerts O, Waer M
机构信息
Laboratory for Experimental Medicine and Endocrinology (LEGENDO), Catholic University of Leuven, Belgium.
出版信息
Transplant Proc. 1995 Feb;27(1):640-1.
PMID:7879129
Abstract
摘要
相似文献
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Induction of mixed bone marrow chimerism as potential therapy for autoimmune (type I) diabetes: experience in the NOD model.
Transplant Proc. 1995 Feb;27(1):640-1.
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Protection against autoimmune diabetes by induction of mixed bone marrow chimerism.通过诱导混合骨髓嵌合体预防自身免疫性糖尿病。
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Protection against autoimmune diabetes in mixed bone marrow chimeras: mechanisms involved.混合骨髓嵌合体中自身免疫性糖尿病的防护：相关机制
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Prevention of diabetes in nonobese diabetic mice by nonmyeloablative allogeneic bone marrow transplantation.通过非清髓性同种异体骨髓移植预防非肥胖糖尿病小鼠的糖尿病
Exp Hematol. 2004 Jun;32(6):579-84. doi: 10.1016/j.exphem.2004.03.007.
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Transplantation of bone marrow genetically engineered to express proinsulin II protects against autoimmune insulitis in NOD mice.经基因工程改造以表达胰岛素原II的骨髓移植可预防非肥胖糖尿病（NOD）小鼠的自身免疫性胰岛炎。
J Gene Med. 2006 Nov;8(11):1281-90. doi: 10.1002/jgm.968.
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Resistance to the induction of mixed chimerism in spontaneously diabetic NOD mice depends on the CD40/CD154 pathway and donor MHC disparity.自发糖尿病NOD小鼠中对混合嵌合体诱导的抗性取决于CD40/CD154途径和供体主要组织相容性复合体差异。
Ann N Y Acad Sci. 2007 Apr;1103:94-102. doi: 10.1196/annals.1394.015. Epub 2007 Mar 21.
9
Low levels of allogeneic but not syngeneic hematopoietic chimerism reverse autoimmune insulitis in prediabetic NOD mice.低水平的异基因而非同基因造血嵌合体可逆转糖尿病前期NOD小鼠的自身免疫性胰岛炎。
J Autoimmun. 2009 Sep;33(2):83-91. doi: 10.1016/j.jaut.2009.07.001. Epub 2009 Jul 24.
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Bone marrow transplantation prevents autoimmune diabetes in nonobese diabetic mice.
Transplant Proc. 1993 Oct;25(5):2896-7.
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