Han D H, Gwak H S, Chung C K
Department of Neurosurgery, Seoul National University College of Medicine, Korea.
Surg Neurol. 1994 Nov;42(5):388-91. doi: 10.1016/0090-3019(94)90341-7.
A case of ruptured aneurysm at the origin of accessory middle cerebral artery associated with ipsilateral middle cerebral artery aplasia is reported. The angiograms did not show a middle cerebral artery shadow on the lesion side. Instead, the accessory middle cerebral artery ran toward the Sylvian fissure and supplied the middle cerebral artery territory along with multiple hypertrophied perforating branches. The operative findings confirmed that the cord-like, rudimentary middle cerebral artery had no internal blood flow. The effect of these combined anomalies on the hemodynamic equilibrium between the arteries of the same embryologic trees and the genesis of aneurysm is worthy of notice.
报告了一例大脑中动脉副干起始部动脉瘤破裂,同时伴有同侧大脑中动脉发育不全的病例。血管造影显示病变侧无大脑中动脉影。相反,大脑中动脉副干走向外侧裂,并与多条增粗的穿支一起供应大脑中动脉供血区。手术所见证实条索状的发育不全大脑中动脉无内部血流。这些联合异常对同一胚胎起源动脉之间血流动力学平衡及动脉瘤发生的影响值得关注。