肌萎缩侧索硬化症：兴奋性毒性是关键所在吗？ - Suppr | 超能文献

Amyotrophic lateral sclerosis: is excitotoxicity the key?

作者信息

Krueger G

出版信息

CMAJ. 1994 Aug 15;151(4):441-3.

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1337096/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6936/1337096/ef1ab8d5af21/cmaj00052-0044-a.jpg

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本文引用的文献

1

Activation of PKC reverses apparent NMDA receptor reduction in ALS.蛋白激酶C的激活可逆转肌萎缩侧索硬化症中明显的N-甲基-D-天冬氨酸受体减少。

Neuroreport. 1993 Jul;4(7):931-4. doi: 10.1097/00001756-199307000-00023.

2

Pathogenic mechanisms in sporadic amyotrophic lateral sclerosis.散发性肌萎缩侧索硬化症的发病机制

Can J Neurol Sci. 1993 Nov;20(4):286-96.

3

A controlled trial of riluzole in amyotrophic lateral sclerosis. ALS/Riluzole Study Group.利鲁唑治疗肌萎缩侧索硬化症的对照试验。肌萎缩侧索硬化症/利鲁唑研究组

N Engl J Med. 1994 Mar 3;330(9):585-91. doi: 10.1056/NEJM199403033300901.