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硬脑膜动静脉畸形所致静脉缺血。病例报告。

Venous ischemia caused by dural arteriovenous malformation. Case report.

作者信息

Kurata A, Miyasaka Y, Yoshida T, Kunii M, Yada K, Kan S

机构信息

Department of Neurosurgery, Kitasato University School of Medicine, Kanagawa, Japan.

出版信息

J Neurosurg. 1994 Mar;80(3):552-5. doi: 10.3171/jns.1994.80.3.0552.

Abstract

A case is presented of tentorial dural arteriovenous malformation (AVM) associated with visual hallucinations and quadrant hemianopsia. Computerized tomography (CT) and magnetic resonance imaging showed an ischemic region, mainly affecting the white matter of the right occipital lobe, that was defined as an area of increased blood volume on dynamic CT scans and as a decrease in cerebral blood flow on N-isopropyl-p-123I-iodoamphetamine single-photon emission CT scans. Angiography demonstrated venous congestion, probably because the retrograde arterial inflow from the dural AVM into the corticomedullary vein was direct and not via the sinuses. The symptoms and radiological findings improved immediately after endovascular treatment. The origin of these symptoms was fully evaluated and confirmed to be a reversible ischemic change caused by disturbance of the volume of venous return over an extensive area.

摘要

本文报告一例天幕硬脑膜动静脉畸形(AVM),伴有视幻觉和象限性偏盲。计算机断层扫描(CT)和磁共振成像显示一个缺血区域,主要累及右侧枕叶白质,在动态CT扫描中该区域被定义为血容量增加区,在N-异丙基-p-123I-碘安非他明单光子发射CT扫描中显示脑血流量减少。血管造影显示静脉淤血,可能是因为硬脑膜AVM的逆行动脉血流直接流入皮质髓质静脉,而非通过静脉窦。血管内治疗后症状和影像学表现立即改善。对这些症状的起源进行了全面评估,证实是由广泛区域静脉回流体积紊乱引起的可逆性缺血改变。

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