Wilson B A, Davidoff J
MRC Applied Psychology Unit, Cambridge.
Cortex. 1993 Sep;29(3):529-42. doi: 10.1016/s0010-9452(13)80258-4.
We describe a young woman, J.R., who sustained a very severe head injury in 1981 at the age of 17 years. She was assessed in 1982 and found to have visual agnosia. Since then J.R. has been assessed on several occasions over a period of ten years. Her agnosia for real objects has resolved and she has improved on the identification of other classes of stimuli. However, she still has some problems with the identification of line drawings, photographs and model animals. Her drawing from memory remains particularly poor and she has difficulty with visual imagery. We consider her residual deficits in the light of Farah's (1990) theoretical framework; this proposes that associative agnosia could be due to a disconnection syndrome, a loss of stored visual representations or to the loss of knowledge of how to perceive objects. J.R.'s residual impairments appear to be mainly due to a loss of access to visual representations in the absence of visual input.
我们描述了一位名叫J.R.的年轻女性，她在1981年17岁时遭受了非常严重的头部损伤。1982年对她进行评估时，发现她患有视觉失认症。从那时起，在十年的时间里J.R.接受了多次评估。她对真实物体的失认症已经消失，并且在识别其他类别的刺激方面有所改善。然而，她在识别线条画、照片和动物模型方面仍然存在一些问题。她凭记忆画画的能力仍然特别差，并且在视觉想象方面存在困难。我们根据法拉（1990年）的理论框架来考虑她的残留缺陷；该理论框架提出，联想性失认症可能是由于连接障碍综合征、存储的视觉表征丧失或对如何感知物体的知识丧失所致。J.R.的残留损伤似乎主要是由于在没有视觉输入的情况下无法获取视觉表征。
