Rothman S A, Miller J M, Hsia H H, Buxton A E
Department of Medicine, Temple University School of Medicine, Philadelphia, Pennsylvania, USA.
J Cardiovasc Electrophysiol. 1995 Jul;6(7):544-50. doi: 10.1111/j.1540-8167.1995.tb00427.x.
Interatrial conduction of recipient atrial tachycardia to the donor atria of an orthotopic heart transplant recipient resulted in a unique cause of supraventricular tachycardia. An electrophysiologic study was performed, and the recipient atria was found to be in an atrial tachycardia, cycle length of 210 msec, with periods of both 2:1 and 1:1 conduction to the donor atria resulting in a donor atrial cycle length of 420 msec and 210 msec, respectively. The site of interatrial conduction was mapped to the right atrial suture line, along the atrial free wall, and was successfully disrupted with radiofrequency energy. Arrhythmias of a similar mechanism may also be observed in other postsurgical patients.
在原位心脏移植受者中,受者房性心动过速经房间传导至供者心房,导致了一种独特的室上性心动过速病因。进行了电生理研究,发现受者心房处于房性心动过速状态,周期长度为210毫秒,存在2:1和1:1传导至供者心房的时期,导致供者心房周期长度分别为420毫秒和210毫秒。房间传导部位定位于右心房缝线处,沿心房游离壁,通过射频能量成功阻断。其他术后患者也可能观察到类似机制的心律失常。