生存还是毁灭：乙醇如何影响发育过程中的神经元死亡。 - Suppr

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To be or not to be: how ethanol can affect neuronal death during development.

作者信息

Hoffman P L, Tabakoff B

机构信息

Department of Pharmacology, University of Colorado, Denver, CO, USA.

出版信息

Alcohol Clin Exp Res. 1996 Feb;20(1):193-5. doi: 10.1111/j.1530-0277.1996.tb01065.x.

DOI:10.1111/j.1530-0277.1996.tb01065.x

PMID:8651453

Abstract

摘要

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To be or not to be: how ethanol can affect neuronal death during development.

作者信息

Hoffman P L, Tabakoff B

机构信息

Department of Pharmacology, University of Colorado, Denver, CO, USA.

出版信息

Alcohol Clin Exp Res. 1996 Feb;20(1):193-5. doi: 10.1111/j.1530-0277.1996.tb01065.x.

DOI:10.1111/j.1530-0277.1996.tb01065.x

PMID:8651453

Abstract

摘要

相似文献

To be or not to be: how ethanol can affect neuronal death during development.

Alcohol Clin Exp Res. 1996 Feb;20(1):193-5. doi: 10.1111/j.1530-0277.1996.tb01065.x.

Mechanisms of ethanol induced neuronal death during development: from the molecule to behavior.

Alcohol Clin Exp Res. 1996 Nov;20(8 Suppl):128A-132A. doi: 10.1111/j.1530-0277.1996.tb01762.x.

Time course and manner of Purkinje neuron death following a single ethanol exposure on postnatal day 4 in the developing rat.

Neuroscience. 2002;114(2):327-37. doi: 10.1016/s0306-4522(02)00344-5.

Intragastric intubation of alcohol during postnatal development of rats results in selective cell loss in the cerebellum.

Alcohol Clin Exp Res. 1993 Dec;17(6):1275-80. doi: 10.1111/j.1530-0277.1993.tb05241.x.

Acid-sensitive channel inhibition prevents fetal alcohol spectrum disorders cerebellar Purkinje cell loss.

Am J Physiol Regul Integr Comp Physiol. 2008 Aug;295(2):R596-603. doi: 10.1152/ajpregu.90321.2008. Epub 2008 May 28.

Alcohol-induced neuroapoptosis in the fetal macaque brain.

Neurobiol Dis. 2010 Oct;40(1):200-6. doi: 10.1016/j.nbd.2010.05.025. Epub 2010 May 23.

A computational model for neocortical neuronogenesis predicts ethanol-induced neocortical neuron number deficits.

Dev Neurosci. 2002;24(6):467-77. doi: 10.1159/000069357.

Neuroanatomical and neurophysiological mechanisms involved in central nervous system dysfunctions induced by prenatal alcohol exposure.

Alcohol Clin Exp Res. 1998 Apr;22(2):304-12. doi: 10.1111/j.1530-0277.1998.tb03653.x.

Ethanol-induced neuroapoptosis in the developing rodent cerebellum and related brain stem structures.

Brain Res Dev Brain Res. 2005 Mar 22;155(1):1-13. doi: 10.1016/j.devbrainres.2004.11.005. Epub 2005 Jan 22.

Effect of pre- or postnatal exposure to ethanol on the total number of neurons in the principal sensory nucleus of the trigeminal nerve: cell proliferation and neuronal death.

Alcohol Clin Exp Res. 1995 Oct;19(5):1359-63. doi: 10.1111/j.1530-0277.1995.tb01625.x.

引用本文的文献

Ethanol induces persistent potentiation of 5-HT receptor-stimulated GABA release at synapses on rat hippocampal CA1 neurons.

Neuropharmacology. 2021 Feb 15;184:108415. doi: 10.1016/j.neuropharm.2020.108415. Epub 2020 Dec 1.

Murine Models for the Study of Fetal Alcohol Spectrum Disorders: An Overview.

Front Pediatr. 2020 Jul 15;8:359. doi: 10.3389/fped.2020.00359. eCollection 2020.

Critical periods of vulnerability for the developing nervous system: evidence from humans and animal models.

Environ Health Perspect. 2000 Jun;108 Suppl 3(Suppl 3):511-33. doi: 10.1289/ehp.00108s3511.