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野生型p53肿瘤抑制基因的表达及其在甲状腺肿瘤细胞凋亡中的可能作用。

Expression of wild-type p53 tumor suppressor gene and its possible involvement in the apoptosis of thyroid tumors.

作者信息

Kikuchi S, Hiraide H, Tamakuma S, Yamamoto M

机构信息

First Department of Surgery, National Defense Medical College, Saitama, Japan.

出版信息

Surg Today. 1997;27(3):226-33. doi: 10.1007/BF00941650.

Abstract

A good prognosis is often achieved in patients who have undergone treatment for human papillary carcinoma of the thyroid. On the assumption that this may be partly attributable to an apoptotic tendency of this special type of tumor, we measured DNA fragmentation, cell death by enzyme-linked immunosorbent assay (ELISA), and the expression of apoptosis-related genes. DNA fragmentation occurred more extensively in malignant tumor cells than in benign thyroid tumors or normal thyroid tissue, as examined by agarose gel electrophoresis and confirmed by the quantitative method using an ELISA kit. Although only expression of the tumor suppressor gene, p53, was increased in the tumor tissue, no expression of other genes, such as Fas, TNF, c-myc, c-fos or bcl-2, was observed in the normal, benign, or malignant tumor tissues, indicating that the roles of these gene functions, if any, were minimal in these tissues. Since p53 is closely related to cellular apoptosis and no point mutation was observed in the transcripts expressed by malignant cells, apoptosis and/or the production of an angiogenesis inhibitor induced by wild-type p53 molecules may be related to the favorable prognosis of patients treated for papillary carcinoma of the thyroid.

摘要

接受过甲状腺乳头状癌治疗的患者通常预后良好。基于这种情况可能部分归因于这种特殊类型肿瘤的凋亡倾向这一假设,我们检测了DNA片段化、通过酶联免疫吸附测定(ELISA)检测细胞死亡情况以及凋亡相关基因的表达。通过琼脂糖凝胶电泳检测并使用ELISA试剂盒定量方法证实,恶性肿瘤细胞中的DNA片段化比良性甲状腺肿瘤或正常甲状腺组织中更为广泛。虽然肿瘤组织中仅肿瘤抑制基因p53的表达增加,但在正常、良性或恶性肿瘤组织中均未观察到其他基因如Fas、TNF、c-myc、c-fos或bcl-2的表达,这表明这些基因功能(如果有的话)在这些组织中的作用极小。由于p53与细胞凋亡密切相关,且在恶性细胞表达的转录本中未观察到点突变,野生型p53分子诱导的细胞凋亡和/或血管生成抑制剂的产生可能与甲状腺乳头状癌治疗患者的良好预后有关。

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