Adair E R, Adams B W, Kelleher S A, Streett J W
John B. Pierce Laboratory, New Haven, Connecticut 06519, USA.
Ann N Y Acad Sci. 1997 Mar 15;813:497-507. doi: 10.1111/j.1749-6632.1997.tb51739.x.
We have examined experimentally the question of increased vulnerability to the thermalizing effects of MW exposure during febrile illness. In a controlled ambient temperature of 26 degrees C, autonomic mechanisms of heat production and heat loss were measured in febrile squirrel monkeys during 30-min exposures to 450 or 2450 MHz CW MW fields at different phases of the fever cycle (induction, plateau, defervescence). We have shown that MW energy absorbed during a febrile episode spares endogenous energy production, but may augment the fever if deposited deep in the body, as is the case during exposure at the resonant frequency. The fever may also be exacerbated if the MW exposure occurs late in the febrile episode, a condition that may put an organism at some risk, especially if the field strength exceeds safety guidelines.
我们通过实验研究了发热疾病期间对微波暴露热效应的易感性增加这一问题。在26摄氏度的受控环境温度下,在发热松鼠猴发热周期的不同阶段(诱导期、平台期、退热期),对其进行30分钟450或2450兆赫连续波微波场暴露,同时测量其产热和散热的自主调节机制。我们已经表明,发热期间吸收的微波能量可节省内源性能量产生,但如果像在共振频率暴露时那样沉积在身体深处,则可能加剧发热。如果在发热后期进行微波暴露,发热也可能加剧,这种情况可能会使生物体面临一定风险,尤其是当场强超过安全准则时。