The leukemic core binding factor beta-smooth muscle myosin heavy chain (CBF beta-SMMHC) chimeric protein requires both CBF beta and myosin heavy chain domains for transformation of NIH 3T3 cells.
作者信息
Adelstein R S, Collins F S, Hajra A, Kelley C A, Liu P P, Speck N A, Stacy T, Wang Q
出版信息
Proc Natl Acad Sci U S A. 1996 Dec 24;93(26):15523.
Abstract
摘要
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1
The leukemic core binding factor beta-smooth muscle myosin heavy chain (CBF beta-SMMHC) chimeric protein requires both CBF beta and myosin heavy chain domains for transformation of NIH 3T3 cells.白血病核心结合因子β-平滑肌肌球蛋白重链（CBFβ-SMMHC）嵌合蛋白在转化NIH 3T3细胞时需要CBFβ和肌球蛋白重链结构域。
Proc Natl Acad Sci U S A. 1996 Dec 24;93(26):15523.
2
The leukemic core binding factor beta-smooth muscle myosin heavy chain (CBF beta-SMMHC) chimeric protein requires both CBF beta and myosin heavy chain domains for transformation of NIH 3T3 cells.白血病核心结合因子β-平滑肌肌球蛋白重链（CBFβ-SMMHC）嵌合蛋白在转化NIH 3T3细胞时需要CBFβ和肌球蛋白重链结构域。
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3
Overexpression of core-binding factor alpha (CBF alpha) reverses cellular transformation by the CBF beta-smooth muscle myosin heavy chain chimeric oncoprotein.核心结合因子α（CBFα）的过表达可逆转由CBFβ-平滑肌肌球蛋白重链嵌合癌蛋白引起的细胞转化。
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The chimeric protein, PEBP2 beta/CBF beta-SMMHC, disorganizes cytoplasmic stress fibers and inhibits transcriptional activation.嵌合蛋白PEBP2β/CBFβ-SMMHC会破坏细胞质应激纤维的结构并抑制转录激活。
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Overexpression of core-binding factor alpha (CBF alpha) reverses cellular transformation by the CBF beta-smooth muscle myosin heavy chain chimeric oncoprotein.核心结合因子α（CBFα）的过表达可逆转由CBFβ-平滑肌肌球蛋白重链嵌合癌蛋白引起的细胞转化。
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Immunohistochemical analysis of CBFbeta-SMMHC protein reveals a unique nuclear localization in acute myeloid leukemia with inv(16)(p13q22).CBFβ-SMMHC蛋白的免疫组织化学分析显示，在伴有inv(16)(p13q22)的急性髓系白血病中存在独特的核定位。
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Age dependence of smooth muscle myosin expression by cultured rat aortic smooth muscle cells.培养的大鼠主动脉平滑肌细胞中平滑肌肌球蛋白表达的年龄依赖性
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