Metabolic and genetic aspects of familial combined hyperlipidaemia with emphasis on low-density lipoprotein heterogeneity.

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作者信息

Bredie S J, Demacker P N, Stalenhoef A F

机构信息

Department of Medicine, University Hospital Nijmegen, The Netherlands.

出版信息

Eur J Clin Invest. 1997 Oct;27(10):802-11. doi: 10.1046/j.1365-2362.1997.1850734.x.

DOI:10.1046/j.1365-2362.1997.1850734.x

PMID:9373757

Abstract

摘要

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Metabolic and genetic aspects of familial combined hyperlipidaemia with emphasis on low-density lipoprotein heterogeneity.

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引用本文的文献

Small and dense LDL in familial combined hyperlipidemia and N291S polymorphism of the lipoprotein lipase gene.家族性混合型高脂血症中的小而密低密度脂蛋白与脂蛋白脂肪酶基因的N291S多态性

Lipids Health Dis. 2009 Mar 31;8:12. doi: 10.1186/1476-511X-8-12.

Metabolic pathogenesis of familial combined hyperlipidaemia with emphasis on insulin resistance, adipose tissue metabolism and free fatty acids.家族性混合性高脂血症的代谢发病机制，重点关注胰岛素抵抗、脂肪组织代谢和游离脂肪酸。

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A genome scan for familial combined hyperlipidemia reveals evidence of linkage with a locus on chromosome 11.一项针对家族性混合性高脂血症的全基因组扫描揭示了与11号染色体上一个位点存在连锁关系的证据。

Am J Hum Genet. 1999 Aug;65(2):397-412. doi: 10.1086/302490.

Families with familial combined hyperlipidemia and families enriched for coronary artery disease share genetic determinants for the atherogenic lipoprotein phenotype.患有家族性混合性高脂血症的家族以及富含冠状动脉疾病的家族共享致动脉粥样硬化脂蛋白表型的遗传决定因素。

Am J Hum Genet. 1998 Aug;63(2):577-85. doi: 10.1086/301983.

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