类风湿关节炎中的细胞凋亡：滑膜组织调节的新途径 - Suppr

Suppr

超能文献

Apoptosis in rheumatoid arthritis: a novel pathway in the regulation of synovial tissue.

作者信息

Nishioka K, Hasunuma T, Kato T, Sumida T, Kobata T

机构信息

St. Marianna University, Kawasaki, Japan.

出版信息

Arthritis Rheum. 1998 Jan;41(1):1-9. doi: 10.1002/1529-0131(199801)41:1<1::AID-ART1>3.0.CO;2-V.

DOI:10.1002/1529-0131(199801)41:1<1::AID-ART1>3.0.CO;2-V

PMID:9433863

Abstract

摘要

相似文献

Apoptosis in rheumatoid arthritis: a novel pathway in the regulation of synovial tissue.类风湿关节炎中的细胞凋亡：滑膜组织调节的新途径

Arthritis Rheum. 1998 Jan;41(1):1-9. doi: 10.1002/1529-0131(199801)41:1<1::AID-ART1>3.0.CO;2-V.

Bone morphogenetic proteins 2 and 6, expressed in arthritic synovium, are regulated by proinflammatory cytokines and differentially modulate fibroblast-like synoviocyte apoptosis.在关节炎滑膜中表达的骨形态发生蛋白2和6受促炎细胞因子调控，并对成纤维样滑膜细胞凋亡进行差异性调节。

Arthritis Rheum. 2003 Oct;48(10):2807-18. doi: 10.1002/art.11389.

Expression of IL-19 and its receptors in RA: potential role for synovial hyperplasia formation.白细胞介素-19及其受体在类风湿性关节炎中的表达：滑膜增生形成的潜在作用。

Rheumatology (Oxford). 2008 Jun;47(6):815-20. doi: 10.1093/rheumatology/ken061. Epub 2008 Apr 8.

Rheumatology (Oxford). 2006 Jan;45(1):26-30. doi: 10.1093/rheumatology/kei113. Epub 2005 Sep 13.

Up-regulation of IL-23p19 expression in rheumatoid arthritis synovial fibroblasts by IL-17 through PI3-kinase-, NF-kappaB- and p38 MAPK-dependent signalling pathways.白细胞介素-17通过磷脂酰肌醇3激酶、核因子κB和p38丝裂原活化蛋白激酶依赖性信号通路上调类风湿关节炎滑膜成纤维细胞中白细胞介素-23p19的表达。

Rheumatology (Oxford). 2007 Jan;46(1):57-64. doi: 10.1093/rheumatology/kel159. Epub 2006 Jun 12.

[Molecular mechanism of Fas signal transduction by synovial cells affected by chronic rheumatoid arthritis].[慢性类风湿关节炎影响下的滑膜细胞Fas信号转导的分子机制]

Nihon Rinsho Meneki Gakkai Kaishi. 1997 Dec;20(6):513-6.

Tumor necrosis factor alpha regulation of the FAS-mediated apoptosis-signaling pathway in synovial cells.肿瘤坏死因子α对滑膜细胞中FAS介导的凋亡信号通路的调节作用。

Arthritis Rheum. 1999 Mar;42(3):519-26. doi: 10.1002/1529-0131(199904)42:3<519::AID-ANR17>3.0.CO;2-Q.

Down-regulation of FLIP sensitizes rheumatoid synovial fibroblasts to Fas-mediated apoptosis.FLIP的下调使类风湿性滑膜成纤维细胞对Fas介导的凋亡敏感。

Arthritis Rheum. 2004 Sep;50(9):2803-10. doi: 10.1002/art.20453.

Potential withdrawal of rheumatoid synovium by the induction of apoptosis using a novel in vivo model of rheumatoid arthritis.使用类风湿性关节炎新型体内模型通过诱导细胞凋亡来实现类风湿滑膜的潜在清除。

Arthritis Rheum. 1998 Jul;41(7):1251-7. doi: 10.1002/1529-0131(199807)41:7<1251::AID-ART16>3.0.CO;2-B.

Cell death in rheumatoid arthritis.类风湿关节炎中的细胞死亡

Apoptosis. 2009 Apr;14(4):447-54. doi: 10.1007/s10495-009-0317-y.

引用本文的文献

Lymphocyte apoptosis and its association with the inflammatory markers and disease severity in juvenile-onset systemic lupus erythematosus patients.淋巴细胞凋亡及其与青少年发病系统性红斑狼疮患者炎症标志物和疾病严重程度的关系。

Pediatr Rheumatol Online J. 2024 Jan 19;22(1):20. doi: 10.1186/s12969-024-00953-9.

A Serum Biomarker Panel of exomiR-451a, exomiR-25-3p and Soluble TWEAK for Early Diagnosis of Rheumatoid Arthritis.外泌体 miR-451a、miR-25-3p 和可溶性 TWEAK 血清生物标志物联合检测对类风湿关节炎的早期诊断价值

Front Immunol. 2021 Nov 15;12:790880. doi: 10.3389/fimmu.2021.790880. eCollection 2021.

Oridonin Inhibits Cell Proliferation and Induces Apoptosis in Rheumatoid Arthritis Fibroblast-Like Synoviocytes.冬凌草甲素抑制类风湿关节炎成纤维样滑膜细胞的增殖并诱导其凋亡。

Inflammation. 2016 Apr;39(2):873-80. doi: 10.1007/s10753-016-0318-2.

Apoptotic Effect of Geniposide on Fibroblast-Like Synoviocytes in Rats with Adjuvant-Induced Arthritis via Inhibiting ERK Signal Pathway In Vitro.栀子苷通过体外抑制ERK信号通路对佐剂性关节炎大鼠成纤维样滑膜细胞的凋亡作用

Inflammation. 2016 Feb;39(1):30-38. doi: 10.1007/s10753-015-0219-9.

Relation of the Fas and FasL gene polymorphisms with susceptibility to and severity of rheumatoid arthritis.Fas 和 FasL 基因多态性与类风湿关节炎易感性和严重程度的关系。

Rheumatol Int. 2013 Oct;33(10):2637-45. doi: 10.1007/s00296-013-2793-1. Epub 2013 Jun 9.

Apoptosis of rheumatoid arthritis fibroblast-like synoviocytes: possible roles of nitric oxide and the thioredoxin 1.类风湿关节炎成纤维样滑膜细胞的凋亡：一氧化氮和硫氧还蛋白 1 的可能作用。

Mediators Inflamm. 2013;2013:953462. doi: 10.1155/2013/953462. Epub 2013 Apr 3.

Gene expression analysis of rheumatoid arthritis synovial lining regions by cDNA microarray combined with laser microdissection: up-regulation of inflammation-associated STAT1, IRF1, CXCL9, CXCL10, and CCL5.通过 cDNA 微阵列结合激光显微切割分析类风湿关节炎滑膜衬里区域的基因表达：炎症相关 STAT1、IRF1、CXCL9、CXCL10 和 CCL5 的上调。

Scand J Rheumatol. 2012 May;41(3):170-9. doi: 10.3109/03009742.2011.623137. Epub 2012 Mar 9.

ARG098, a novel anti-human Fas antibody, suppresses synovial hyperplasia and prevents cartilage destruction in a severe combined immunodeficient-HuRAg mouse model.ARG098，一种新型抗人 Fas 抗体，可抑制滑膜过度增生并预防严重联合免疫缺陷-HuRAg 小鼠模型中的软骨破坏。

BMC Musculoskelet Disord. 2010 Sep 27;11:221. doi: 10.1186/1471-2474-11-221.

Changes in apoptotic gene expression in lymphocytes from rheumatoid arthritis and systemic lupus erythematosus patients compared with healthy lymphocytes.与健康淋巴细胞相比，类风湿关节炎和系统性红斑狼疮患者淋巴细胞中凋亡基因表达的变化。

J Clin Immunol. 2010 Sep;30(5):649-58. doi: 10.1007/s10875-010-9429-y. Epub 2010 Jun 9.

The contribution of Asian researchers to the field of rheumatology.亚洲研究者对风湿病学领域的贡献。

Nat Rev Rheumatol. 2010 Feb;6(2):106-11. doi: 10.1038/nrrheum.2009.257.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验