Patole S, Whitehall J, Almonte R, Stalewski H, Lee-Tannock A, Murphy A
Department of Neonatology, Kirwan Hospital for Women, Townsville, Queensland, Australia.
Am J Perinatol. 1998 Jan;15(1):53-6. doi: 10.1055/s-2007-993899.
A case is presented in which extension of meconium peritonitis through muscular defects in the diaphragm lead to intrathoracic calcifications diagnosed sonographically at 23 weeks of gestation. There were three diaphragmatic defects, two small ones corresponded to foramina of Morgagni and one large posterior defect that did not correspond to the foramen of Bochdelak. There were three additional muscular defects: one in the rectus abdominus and two, bilaterally, in the loins. Despite long-standing fetal ascites and fresh intraperitoneal meconium at laparotomy, postoperative progress was uneventful. The baby did not have other dysmorphic features except for a single palmar crease, the chromosomes were normal, and the baby did not have cystic fibrosis. Follow-up examination at 10 months showed a thriving infant with mild hypotonia and developmental delay, but no respiratory or gastrointestinal problems.
本文报告了一例病例,胎粪性腹膜炎通过膈肌的肌肉缺损蔓延至胸腔,在妊娠23周时经超声诊断为胸腔内钙化。存在三处膈肌缺损,两处小缺损对应于莫尔加尼孔,一处大的后部缺损不对应于博赫德莱克孔。另外还有三处肌肉缺损:一处在腹直肌,两处位于双侧腰部。尽管剖腹手术时发现长期存在胎儿腹水和新鲜的腹腔内胎粪,但术后恢复顺利。除了单一掌纹外,婴儿没有其他畸形特征,染色体正常,且婴儿没有囊性纤维化。10个月时的随访检查显示婴儿发育良好,有轻度肌张力减退和发育迟缓,但没有呼吸或胃肠道问题。