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编码Not蛋白复合体新组分的NOT5的特性分析。

Characterization of NOT5 that encodes a new component of the Not protein complex.

作者信息

Oberholzer U, Collart M A

机构信息

Department of Medical Biochemistry, CMU, Geneva, Switzerland.

出版信息

Gene. 1998 Jan 19;207(1):61-9. doi: 10.1016/s0378-1119(97)00605-7.

Abstract

The yeast HIS3 gene has two core promoters: TC, a TATA-less element and TR, a canonical TATA element. Four genes encode global negative regulators of transcription that preferentially repress TC-dependent transcription: NOT1 (CDC39), NOT2 (CDC36), NOT3 and NOT4 (SIG1, MOT2). Genetic and biochemical experiments suggest that the products of these genes are associated in a complex and regulate TFIID function. In this paper, we describe a new gene, NOT5, that also represses transcription of the HIS3 TATA-less promoter preferentially and encodes a protein whose N-terminal region is 44% identical to that of Not3p. Our results indicate that NOT5 is involved in Not function and encodes a product that is physically associated with the other Not proteins. First, overexpression of NOT3 or NOT4 suppresses mutations in NOT5. Secondly, mutations in NOT4 are synthetically lethal with mutations in NOT5. Thirdly, NOT5 interacts with NOT1 and NOT3 in the two-hybrid assay. Finally, Not1p, Not3p and Not4p co-immunoprecipitate with Not5p.

摘要

酵母HIS3基因有两个核心启动子:TC,一个无TATA元件;TR,一个典型的TATA元件。四个基因编码转录的全局负调控因子,它们优先抑制依赖TC的转录:NOT1(CDC39)、NOT2(CDC36)、NOT3和NOT4(SIG1、MOT2)。遗传和生化实验表明,这些基因的产物以复合物形式存在并调节TFIID功能。在本文中,我们描述了一个新基因NOT5,它也优先抑制HIS3无TATA启动子的转录,并编码一种蛋白质,其N端区域与Not3p的N端区域有44%的同源性。我们的结果表明,NOT5参与Not功能,并编码一种与其他Not蛋白存在物理关联的产物。首先,NOT3或NOT4的过表达可抑制NOT5中的突变。其次,NOT4中的突变与NOT5中的突变是合成致死的。第三,在双杂交实验中,NOT5与NOT1和NOT3相互作用。最后,Not1p、Not3p和Not4p与Not5p共免疫沉淀。

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