牛高分子量激肽原及其片段对菲茨杰拉德性状血浆的影响。 - Suppr

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超能文献

Effect of bovine high molecular weight kininogen and its fragments on Fitzgerald trait plasma.

作者信息

Waldmann R, Scicli A G, McGregor R K, Carretero O A, Abraham J P, Kato H, Han Y N, Iwanaga S

出版信息

Thromb Res. 1976 Jun;8(6):785-95. doi: 10.1016/0049-3848(76)90007-4.

DOI:10.1016/0049-3848(76)90007-4

PMID:960054

Abstract

摘要

相似文献

Effect of bovine high molecular weight kininogen and its fragments on Fitzgerald trait plasma.牛高分子量激肽原及其片段对菲茨杰拉德性状血浆的影响。

Thromb Res. 1976 Jun;8(6):785-95. doi: 10.1016/0049-3848(76)90007-4.

Kininogen deficiency in Fitzgerald trait: role of high molecular weight kininogen in clotting and fibrinolysis.菲茨杰拉德性状中的激肽原缺乏：高分子量激肽原在凝血和纤维蛋白溶解中的作用。

J Lab Clin Med. 1976 Feb;87(2):327-37.

Potentiation of the function of Hageman factor fragments by high molecular weight kininogen.高分子量激肽原对哈格曼因子片段功能的增强作用。

J Clin Invest. 1977 Jul;60(1):7-17. doi: 10.1172/JCI108770.

Relation between structure and correcting activity of bovine high molecular weight kininogen upon the clotting time of Fitzgerald-trait plasma.牛高分子量激肽原的结构与对菲茨杰拉德特性血浆凝血时间的校正活性之间的关系。

J Exp Med. 1979 Apr 1;149(4):847-55. doi: 10.1084/jem.149.4.847.

Interactions among Hageman factor (HG, Factor XII), plasma thromboplastin antecedent (PTA, Factor XI), plasma prekallikrein (PK, Fletcher factor) and high molecular weight kininogen (HMW-K, Fitzgerald factor) in blood coagulation.血液凝固过程中，接触因子（HG，凝血因子XII）、血浆凝血活酶前体（PTA，凝血因子XI）、血浆前激肽释放酶（PK，弗莱彻因子）和高分子量激肽原（HMW-K，菲茨杰拉德因子）之间的相互作用。

Adv Exp Med Biol. 1979;120B:61-70.

[Blood coagulation mechanism from the viewpoint of molecular biochemistry: Initiation of blood coagulation].

Rinsho Byori. 1985 Sep;33(9):960-5.

Abnormalities in the contact activation through factor XII in Fujiwara trait: a deficiency in both high and low molecular weight kininogens with low level of prekallikrein.藤原特质中通过因子 XII 的接触激活异常：高分子量和低分子量激肽原均缺乏，且前激肽释放酶水平较低。

Tohoku J Exp Med. 1981 Jan;133(1):67-80. doi: 10.1620/tjem.133.67.

Prekallikrein deficiency in a kindred with kininogen deficiency and Fitzgerald trait clotting defect. Evidence that high molecular weight kininogen and prekallikrein exist as a complex in normal human plasma.一个家族中激肽释放酶原缺乏伴激肽原缺乏和菲茨杰拉德特征性凝血缺陷。证据表明高分子量激肽原和激肽释放酶原在正常人血浆中以复合物形式存在。

J Clin Invest. 1977 Sep;60(3):571-83. doi: 10.1172/JCI108809.

Chloroform-induced fibrinolysis. Its dependence on Hageman factor, plasma prekallikrein, and high molecular weight kininogen.氯仿诱导的纤维蛋白溶解。其对哈格曼因子、血浆前激肽释放酶和高分子量激肽原的依赖性。

J Lab Clin Med. 1980 Apr;95(4):507-14.

Role of HMW kininogen in surface-mediated activation of Factor XII.高分子量激肽原在因子XII表面介导激活中的作用。

Adv Exp Med Biol. 1983;156:73-85.

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J Thromb Haemost. 2008 Nov;6(11):1876-83. doi: 10.1111/j.1538-7836.2008.03143.x. Epub 2008 Aug 28.

Surface activation of factor XII (Hageman factor)--critical role of high molecular weight kininogen and another potentiator).因子 XII（哈格曼因子）的表面激活——高分子量激肽原和另一种增强剂的关键作用。

Agents Actions. 1978 Jan;8(1-2):65-72. doi: 10.1007/BF01972404.

J Exp Med. 1979 Apr 1;149(4):847-55. doi: 10.1084/jem.149.4.847.

The renal kallikrein-kinin system in human and in experimental hypertension.人类及实验性高血压中的肾激肽释放酶-激肽系统

Klin Wochenschr. 1978;56 Suppl 1:113-25. doi: 10.1007/BF01477462.

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