Tyrdal S, Olsen B S
Orthopaedic Department, Ullevaal Hospital, University of Oslo, Norway.
J Shoulder Elbow Surg. 1998 May-Jun;7(3):272-83. doi: 10.1016/s1058-2746(98)90056-8.
According to an epidemiologic study (Scand J Med Sci 1996/ 6: 297-302) the mechanism of "handball goalie's elbow" may be forced hyperextension. The pathomechanics of hyperextension were studied in nine macroscopically normal male cadaver elbow joints. The mean age of the donors was 43.2 years (range 25 to 61 years). Kinematic tests were performed with an experimental three-dimensional kinematic loading apparatus. Hyperextension loads induced joint laxity during flexion of less than 50 degrees. The kinematic changes were significant in joint flexion during forced valgus and external and internal axial rotation, but were not significant in flexion during forced varus. No instability was found with flexion beyond 90 degrees. The hyperextension loads produced four lesions: (1) anterior capsule rupture; (2) L-shaped rupture of the pronator/flexor origin with elongation of the anterior part of the medial collateral ligament; (3) occasional incomplete rupture of the lateral collateral ligament; and (4) small fragments of cartilage near the posterior edge of the ulna in one of the specimens. One or more of these lesions may be responsible for the symptoms in "handball goalie's elbow."
根据一项流行病学研究(《Scand J Med Sci》1996/6:297 - 302),“手球守门员肘”的机制可能是强迫性过度伸展。在9个宏观上正常的男性尸体肘关节中研究了过度伸展的病理力学。供体的平均年龄为43.2岁(范围25至61岁)。使用实验性三维运动加载装置进行运动学测试。在小于50度的屈曲过程中,过度伸展负荷会导致关节松弛。在强迫外翻以及外旋和内旋过程中,关节屈曲时的运动学变化显著,但在强迫内翻时屈曲过程中的变化不显著。屈曲超过90度时未发现不稳定情况。过度伸展负荷产生了四种损伤:(1)前关节囊破裂;(2)旋前肌/屈肌起点的L形破裂,伴有内侧副韧带前部伸长;(3)外侧副韧带偶尔不完全破裂;(4)在其中一个标本中,尺骨后缘附近有小的软骨碎片。这些损伤中的一种或多种可能是“手球守门员肘”症状的原因。