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实验性小鼠刚果锥虫感染。II. 脾脏黏附性CD3⁺Thy1.2⁺TCR-αβ⁻γδ⁻CD4⁺8⁻和CD3⁺Thy1.2⁺TCR-αβ⁻γδ⁻CD4⁻8⁻细胞在白细胞介素-4、白细胞介素-10和干扰素-γ产生以及锥虫引发的免疫抑制中的作用

Experimental murine Trypanosoma congolense infections. II. Role of splenic adherent CD3+Thy1.2+ TCR-alpha beta- gamma delta- CD4+8- and CD3+Thy1.2+ TCR-alpha beta- gamma delta- CD4-8- cells in the production of IL-4, IL-10, and IFN-gamma and in trypanosome-elicited immunosuppression.

作者信息

Uzonna J E, Kaushik R S, Zhang Y, Gordon J R, Tabel H

机构信息

Department of Veterinary Microbiology, Western College of Veterinary Medicine, University of Saskatchewan, Saskatoon, Canada.

出版信息

J Immunol. 1998 Dec 1;161(11):6189-97.

PMID:9834105
Abstract

Trypanosome-induced suppression of T and B cell responses to parasite-related and -unrelated Ags is considered a major mechanism of evasion of the host's immune defenses by the parasite. Reduced T and B cell responses have been attributed to suppressor T cells, suppressor macrophages, or both. We have previously shown that endogenously produced IL-10 and IFN-gamma mediate the suppression of T cell responses in Trypanosoma congolense-infected mice. Here, we show for the first time that splenic CD3+ Thy1.2+ alphabeta- gammadelta- CD4+ 8- and CD3+ Thy1.2+ alphabeta- gammadetla- CD4- 8- cells that copurify with plastic-, nylon wool-, or Sephadex G-10-adherent cell populations, in synergy with adherent Thy1.2- cells, are the major producers of IL-4, IL-10, and IFN-gamma in T. congolense-infected mice. We further demonstrate the involvement of these cells in the suppression of T cell proliferative responses to mitogen and in B cell responses to a parasite-unrelated Ag.

摘要

锥虫诱导的对寄生虫相关和无关抗原的T细胞和B细胞反应抑制被认为是寄生虫逃避宿主免疫防御的主要机制。T细胞和B细胞反应降低归因于抑制性T细胞、抑制性巨噬细胞或两者。我们之前已经表明,内源性产生的白细胞介素-10(IL-10)和干扰素-γ(IFN-γ)介导了冈比亚锥虫感染小鼠中T细胞反应的抑制。在此,我们首次表明,与塑料、尼龙毛或葡聚糖G-10黏附细胞群体共纯化的脾脏CD3⁺Thy1.2⁺αβ⁻γδ⁻CD4⁺8⁻和CD3⁺Thy1.2⁺αβ⁻γδ⁻CD4⁻8⁻细胞,与黏附的Thy1.2⁻细胞协同作用,是冈比亚锥虫感染小鼠中IL-4、IL-10和IFN-γ的主要产生者。我们进一步证明了这些细胞参与对有丝分裂原的T细胞增殖反应抑制以及对寄生虫无关抗原的B细胞反应。

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