Filler R M, Rossello P J, Lebowitz R L
J Pediatr Surg. 1976 Oct;11(5):739-48. doi: 10.1016/0022-3468(76)90098-1.
Three infants who developed anoxic spells 2, 5, and 20 mo following repair of esophageal atresia developed apneic spells during or within a few minutes of feeding. These episodes began with stridor and cyanosis; when severe, they progressed to apnea and loss of consciousness. Mouth-to-mouth resuscitation was often necessary. Investigations failed to detect esophageal obstruction and/or a recurrent tracheoesophageal fistula. No neurologic or cardiac abnormalities were found. The cause was compression of a 1- to 3-cm segment of trachea anteriorly by a vascular structure and posteriorly by a dilated esophagus that emptied slowly because of poor motility. Endoscopy confirmed the x-ray findings. The aortic arch and innominate artery were suspended to the sternum anteriorly, which relieved the apneic spells in all patients.
三名在食管闭锁修复术后2个月、5个月和20个月出现缺氧发作的婴儿,在喂食期间或喂食后几分钟内出现呼吸暂停发作。这些发作始于喘鸣和发绀;严重时,会发展为呼吸暂停和意识丧失。通常需要进行口对口复苏。检查未发现食管梗阻和/或复发性气管食管瘘。未发现神经或心脏异常。病因是一段1至3厘米长的气管前部被一个血管结构压迫,后部被一个因蠕动不良而排空缓慢的扩张食管压迫。内镜检查证实了X线检查结果。主动脉弓和无名动脉向前悬吊至胸骨,这缓解了所有患者的呼吸暂停发作。