Chikugo F, Kitagawa T, Hori T, Masuda Y, Kawahito T, Katoh I
Department of Cardiovascular Surgery, School of Medicine, University of Tokushima, Japan.
Jpn J Thorac Cardiovasc Surg. 1998 Dec;46(12):1329-33. doi: 10.1007/BF03217924.
We experienced a case of a 38-year-old woman with a persistent cough, 24 years after repair of absent pulmonary valve syndrome with anomalous origin of the left pulmonary artery. The right pulmonary artery was massively dilated, thus it caused the compression of the bronchi, which was thought to result in her respiratory symptom. This dilatation of the right pulmonary artery seemed to have progressed because of the following two reasons. The first is the pulmonary hypertension caused by the late reconstruction of the left pulmonary artery. The second is residual pulmonary stenosis and regurgitation after the initial operation without a pulmonary valve insertion. We performed a reoperation consisting of reconstruction of the right ventricular outflow tract using a valved conduit and plication of the right pulmonary artery. Her postoperative course has been without any complications and satisfactory for the past 2 years.
我们遇到了一例38岁女性患者,在修复肺动脉瓣缺如综合征合并左肺动脉异常起源24年后出现持续性咳嗽。右肺动脉明显扩张,从而压迫支气管,这被认为是导致其呼吸道症状的原因。右肺动脉的这种扩张似乎由于以下两个原因而进展。第一个原因是左肺动脉晚期重建导致的肺动脉高压。第二个原因是初次手术未植入肺动脉瓣后残留的肺动脉狭窄和反流。我们进行了再次手术,包括使用带瓣管道重建右心室流出道和折叠右肺动脉。在过去的2年里,她的术后病程没有任何并发症,情况令人满意。