Prolonged anti-CD40 ligand therapy improves primate cardiac allograft survival.
作者信息
Chang A C, Blum M G, Blair K S, Scott M A, Brock J E, Thomas D W, Burkly L C, Miller G G, Pierson R N
机构信息
Vanderbilt University Medical Center, Nashville, Tennessee, USA.
出版信息
Transplant Proc. 1999 Feb-Mar;31(1-2):95. doi: 10.1016/s0041-1345(98)01455-9.
Abstract
摘要
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Prolonged anti-CD40 ligand therapy improves primate cardiac allograft survival.
Transplant Proc. 1999 Feb-Mar;31(1-2):95. doi: 10.1016/s0041-1345(98)01455-9.
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Host CD40 ligand deficiency induces long-term allograft survival and donor-specific tolerance in mouse cardiac transplantation but does not prevent graft arteriosclerosis.宿主CD40配体缺陷可诱导小鼠心脏移植中长期移植物存活和供体特异性耐受，但不能预防移植血管硬化。
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Prolonged blockade of CD40-CD40 ligand interactions by gene transfer of CD40Ig results in long-term heart allograft survival and donor-specific hyporesponsiveness, but does not prevent chronic rejection.通过CD40Ig基因转移对CD40 - CD40配体相互作用进行长期阻断可导致心脏同种异体移植长期存活和供体特异性低反应性，但不能预防慢性排斥反应。
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6
Prolonged skin allograft survival in mice treated with Flt3-ligand-induced dendritic cells and anti-CD154 monoclonal antibody.
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CD40 ligand-CD40 T cell costimulation pathway is required for host sensitization in the immune cascade leading to accelerated allograft rejection.CD40配体-CD40 T细胞共刺激途径是免疫级联反应中宿主致敏所必需的，该反应会导致加速移植排斥反应。
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9
CD40-gp39 interactions play a critical role during allograft rejection. Suppression of allograft rejection by blockade of the CD40-gp39 pathway.CD40与gp39的相互作用在同种异体移植排斥反应中起关键作用。通过阻断CD40-gp39途径抑制同种异体移植排斥反应。
Transplantation. 1996 Jan 15;61(1):4-9. doi: 10.1097/00007890-199601150-00002.
10
CD40-CD40 ligand-independent activation of CD8+ T cells can trigger allograft rejection.CD8+ T细胞的CD40 - CD40配体非依赖性激活可引发同种异体移植排斥反应。
J Immunol. 2000 Jul 15;165(2):1111-8. doi: 10.4049/jimmunol.165.2.1111.
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