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Periodontal immune responses of human lymphocytes in Actinobacillus actinomycetemcomitans-inoculated NOD/SCID mice engrafted with peripheral blood leukocytes of periodontitis patients.

作者信息

Teng Y T, Nguyen H, Hassanloo A, Ellen R P, Hozumi N, Gorczynski R M

机构信息

Department of Microbiology and Immunology, Faculty of Medicine and Dentistry, the University of Western Ontario, London, Canada.

出版信息

J Periodontal Res. 1999 Jan;34(1):54-61. doi: 10.1111/j.1600-0765.1999.tb02222.x.

DOI:10.1111/j.1600-0765.1999.tb02222.x
PMID:10086887
Abstract
摘要

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1
Periodontal immune responses of human lymphocytes in Actinobacillus actinomycetemcomitans-inoculated NOD/SCID mice engrafted with peripheral blood leukocytes of periodontitis patients.接种伴放线放线杆菌的NOD/SCID小鼠移植牙周炎患者外周血白细胞后的人淋巴细胞牙周免疫反应
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Front Immunol. 2021 Jun 17;12:663328. doi: 10.3389/fimmu.2021.663328. eCollection 2021.
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Transient Expression of IL-17A in Foxp3 Fate-Tracked Cells in -Mediated Oral Dysbiosis.IL-17A 在 介导的口腔生态失调中 Foxp3 命运追踪细胞的瞬时表达。
Front Immunol. 2020 Apr 23;11:677. doi: 10.3389/fimmu.2020.00677. eCollection 2020.
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Relevance of Caspase-1 and Nlrp3 Inflammasome on Inflammatory Bone Resorption in A Murine Model of Periodontitis.
Caspase-1 和 Nlrp3 炎性小体在牙周炎小鼠模型中炎症性骨吸收中的相关性。
Sci Rep. 2020 May 8;10(1):7823. doi: 10.1038/s41598-020-64685-y.
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Mucosal Langerhans Cells Promote Differentiation of Th17 Cells in a Murine Model of Periodontitis but Are Not Required for Porphyromonas gingivalis-Driven Alveolar Bone Destruction.黏膜朗格汉斯细胞在牙周炎小鼠模型中促进Th17细胞分化,但不是牙龈卟啉单胞菌驱动的牙槽骨破坏所必需的。
J Immunol. 2016 Aug 15;197(4):1435-46. doi: 10.4049/jimmunol.1502693. Epub 2016 Jul 11.
5
The use of rodent models to investigate host-bacteria interactions related to periodontal diseases.使用啮齿动物模型来研究与牙周疾病相关的宿主-细菌相互作用。
J Clin Periodontol. 2008 Feb;35(2):89-105. doi: 10.1111/j.1600-051X.2007.01172.x.
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Interleukin-10 inhibits gram-negative-microbe-specific human receptor activator of NF-kappaB ligand-positive CD4+-Th1-cell-associated alveolar bone loss in vivo.白细胞介素-10在体内可抑制革兰氏阴性微生物特异性的、核因子κB受体活化因子配体阳性的CD4+ Th1细胞相关的牙槽骨吸收。
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