Potapov A A, Potapova Z G, Medvedev M A
Fiziol Zh SSSR Im I M Sechenova. 1976 Nov;62(11):1691-7.
In chronical experiments on dogs, blocking of glococorticoidal function of adrenal cortex O, pI -- DDD decreases the gall bladder motility. Disorders of mineralcorticoid activity of adrenal cortex leads to an increase of the motility. Administration of hydrocortisone and 1% solution of sodium chloride aids to normalize the gall bladder motility in animals with adrenal insufficiency.
在对狗进行的慢性实验中，肾上腺皮质的糖皮质激素功能被O，pI - DDD阻断会降低胆囊运动性。肾上腺皮质盐皮质激素活性紊乱会导致运动性增加。给予氢化可的松和1%氯化钠溶液有助于使肾上腺功能不全动物的胆囊运动性恢复正常。
