Barton E D
Department of Emergency Medicine, Brigham & Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA.
Curr Opin Pulm Med. 1999 Jul;5(4):269-74. doi: 10.1097/00063198-199907000-00016.
The diagnosis of tension pneumothorax has typically been taught as the presence of hemodynamic compromise with an expanding intrapleural space air mass. This may occur quickly or gradually, depending on the degree of lung injury and respiratory state of the patient. Experimentally, tension pneumothorax is a multifactorial event that manifests a state of central hypoxemia, compensatory mechanisms, and mechanical compression on intrathoracic structures. Studies using animal models suggest that over hypotension is a delayed finding that immediately precedes cardiorespiratory collapse. Recognition of early signs and symptoms associated with tension pneumothorax, e.g., progressive hypoxemia, tachycardia, and respiratory distress, can alert medical personnel to the need for rapid decompression before physiologic decompensation.
典型的张力性气胸诊断依据一直是存在血流动力学障碍以及胸腔内气体量不断增加。这可能迅速或逐渐发生,具体取决于肺损伤程度和患者的呼吸状态。在实验中,张力性气胸是一个多因素事件,表现为中枢性低氧血症、代偿机制以及对胸腔内结构的机械性压迫状态。使用动物模型的研究表明,严重低血压是心肺功能衰竭前的一个延迟表现。识别与张力性气胸相关的早期体征和症状,如进行性低氧血症、心动过速和呼吸窘迫,可提醒医务人员在生理失代偿前需要迅速进行减压。