Harrison M J
Middlesex Hospital, London, UK.
Postgrad Med J. 1999 Mar;75(881):145-6. doi: 10.1136/pgmj.75.881.145.
Three patients are described in whom one or more carotid territory transient ischaemic attacks (TIAs) were preceded by sneezing, straining, or bending over. It is argued that the mechanism involved dislodgment of embolic material from the site of carotid atheroma. This mechanism should be considered as an alternative to paradoxical embolism when TIAs are precipitated by such physiological manoeuvres. Furthermore, TIAs should be added to the list of medical hazards associated with such events.
本文描述了3例患者，他们在一次或多次颈动脉供血区短暂性脑缺血发作（TIA）之前出现了打喷嚏、用力或弯腰的情况。据认为，其机制是颈动脉粥样硬化部位的栓子物质发生脱落。当TIA由这些生理动作诱发时，该机制应被视为反常栓塞的一种替代解释。此外，TIA应被列入与此类事件相关的医学风险清单中。
