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由用力、弯腰和打喷嚏诱发的与颈动脉狭窄相关的短暂性脑缺血发作。

Transient ischaemic attacks related to carotid stenosis precipitated by straining, bending, and sneezing.

作者信息

Harrison M J

机构信息

Middlesex Hospital, London, UK.

出版信息

Postgrad Med J. 1999 Mar;75(881):145-6. doi: 10.1136/pgmj.75.881.145.

Abstract

Three patients are described in whom one or more carotid territory transient ischaemic attacks (TIAs) were preceded by sneezing, straining, or bending over. It is argued that the mechanism involved dislodgment of embolic material from the site of carotid atheroma. This mechanism should be considered as an alternative to paradoxical embolism when TIAs are precipitated by such physiological manoeuvres. Furthermore, TIAs should be added to the list of medical hazards associated with such events.

摘要

本文描述了3例患者,他们在一次或多次颈动脉供血区短暂性脑缺血发作(TIA)之前出现了打喷嚏、用力或弯腰的情况。据认为,其机制是颈动脉粥样硬化部位的栓子物质发生脱落。当TIA由这些生理动作诱发时,该机制应被视为反常栓塞的一种替代解释。此外,TIA应被列入与此类事件相关的医学风险清单中。

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本文引用的文献

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Sneeze-induced hemiparesis.喷嚏诱发的偏瘫。
Ann Neurol. 1982 Jan;11(1):105-6. doi: 10.1002/ana.410110122.
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Cough fracture of the ribs.肋骨咳嗽性骨折
Am J Emerg Med. 1984 Nov;2(6):513-7. doi: 10.1016/0735-6757(84)90077-9.
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Potential sources of emboli in patients with presumed transient cerebral or retinal ischaemia.
Lancet. 1981 Feb 14;1(8216):343-6. doi: 10.1016/s0140-6736(81)91669-x.
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Pulmonary host defense mechanisms.
Chest. 1985 Feb;87(2):247-52. doi: 10.1378/chest.87.2.247.

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