Calò L, Riccardi R, Scaglione M, Caponi D, Golia P, Gaita F
Division of Cardiology, Hospital of Asti.
G Ital Cardiol. 1999 Nov;29(11):1318-22.
Generally, the induction of typical atrioventricular nodal reentrant tachycardia (AVNRT) occurs with a premature atrial stimulus that blocks in the fast pathway and proceeds down the slow pathway slowly enough to allow the refractory fast pathway time to recover. We describe two cases in which a typical AVNRT was induced in an unusual fashion.
The first case is a 41-year-old man with paroxysmal supraventricular tachycardia. During the electrophysiology study, the atrial extrastimulus inducing the typical AVNRT was conducted simultaneously over the fast (AH) and the slow pathway (AH'). A successful ablation of the slow pathway was performed. During the follow-up no recurrence was noted. The second case is a 52-year-old woman with a Wolff-Parkinson-White syndrome due to a left posterior accessory pathway. After 5 minutes of atrioventricular reentrant tachycardia (AVRT) induced by a ventricular extrastimulus, a variability of the antegrade conduction was noted in presence of the same VA conduction. In fact, a short AH interval (fast pathway) alternated with a more prolonged AH intervals (slow pathway) that progressively lengthened until a typical AVNRT was induced. The ablation of the accessory pathway eliminated both tachycardias.
A rare manifestation of dual atrioventricular nodal pathways is a double ventricular response to an atrial impulse that may cause a tachycardia with an atrioventricular conduction of 1:2. In our first case, an atrial extrastimulus was simultaneously conducted over the fast and the slow pathway inducing an AVNRT. This nodal reentry implies two different mechanisms: 1) a retrograde block on the slow pathway impeding the activation of the slow pathway from the impulse coming down the fast pathway, and 2) a critical slowing of conduction in the slow pathway to allow the recovery of excitability of the fast pathway. Interestingly, in the second case, during an AVRT the atrial impulse suddenly proceeded alternately over the fast and the slow pathway. The progressive slowing of conduction over the slow pathway until a certain point which allows the recovery of excitability of the fast pathway determines the AVNRT. This is a case of "tachycardia-induced tachycardia" as confirmed by the fact that the ablation of the accessory pathway eliminated both tachycardias.
一般来说,典型房室结折返性心动过速(AVNRT)的诱发是由于房性期前刺激在快径路发生阻滞,并沿慢径路缓慢下传,使得处于不应期的快径路有足够时间恢复兴奋性。我们描述了两例以不寻常方式诱发典型AVNRT的病例。
第一例为一名41岁阵发性室上性心动过速男性患者。在电生理研究过程中,诱发典型AVNRT的房性期外刺激同时经快径路(AH)和慢径路(AH')下传。成功进行了慢径路消融。随访期间未发现复发。第二例为一名52岁因左侧后间隔旁路导致预激综合征的女性患者。在心室期外刺激诱发房室折返性心动过速(AVRT)5分钟后,在相同室房传导情况下,前传传导出现变化。实际上,短AH间期(快径路)与更长的AH间期(慢径路)交替出现,且后者逐渐延长,直至诱发典型AVNRT。旁路消融消除了两种心动过速。
房室结双径路的一种罕见表现是心房冲动引起双心室反应,这可能导致房室传导比例为1:2的心动过速。在我们的第一例病例中,房性期外刺激同时经快径路和慢径路下传,诱发了AVNRT。这种结内折返意味着两种不同机制:1)慢径路上的逆向阻滞,阻止了来自快径路下传冲动对慢径路的激动;2)慢径路传导显著减慢,以使快径路恢复兴奋性。有趣的是,在第二例病例中,在AVRT期间,心房冲动突然交替经快径路和慢径路下传。慢径路传导逐渐减慢,直至达到某一点,使快径路恢复兴奋性,从而诱发AVNRT。这是一例“心动过速诱发的心动过速”,因为旁路消融消除了两种心动过速,这一事实证实了这一点。