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[I/D polymorphism of the ACE gene and A1166C of the AT1R gene as risk factors for restenosis after coronary angioplasty].

作者信息

Gensini F, Battaglini B, Fatini C, Guazzelli R, Falai M, Chioccioli M, Simonetti I, Comeglio M, Giglioli C, Martinucci P, Gensini G F

机构信息

Unità di Genetica Medica, Università degli Studi, Firenze.

出版信息

Minerva Cardioangiol. 1999 Dec;47(12):516.

PMID:10670180
Abstract
摘要

相似文献

1
[I/D polymorphism of the ACE gene and A1166C of the AT1R gene as risk factors for restenosis after coronary angioplasty].
Minerva Cardioangiol. 1999 Dec;47(12):516.
2
The angiotensinogen gene 235T variant is associated with an increased risk of restenosis after percutaneous transluminal coronary angioplasty.血管紧张素原基因235T变异与经皮腔内冠状动脉成形术后再狭窄风险增加相关。
Clin Sci (Lond). 2000 Jul;99(1):19-25.
3
ACE gene polymorphism and coronary restenosis.血管紧张素转换酶基因多态性与冠状动脉再狭窄
Semin Interv Cardiol. 1999 Sep;4(3):145-9. doi: 10.1053/siic.1999.0085.
4
Renin-angiotensin system gene polymorphisms: assessment of the risk of coronary heart disease.肾素-血管紧张素系统基因多态性：冠心病风险评估
Kardiol Pol. 2003 Jan;58(1):1-9.
5
The ACE gene I/D polymorphism, but not the angiotensin II type I receptor gene A1166C polymorphism is associated with isolated systolic hypertension.
J Hum Hypertens. 2001 Sep;15(9):653-4. doi: 10.1038/sj.jhh.1001221.
6
Angiotensin converting enzyme DD genotype affects the changes of plasma plasminogen activator inhibitor-1 activity after primary percutaneous transluminal coronary angioplasty in acute myocardial infarction patients.血管紧张素转换酶DD基因型影响急性心肌梗死患者直接经皮冠状动脉腔内血管成形术后血浆纤溶酶原激活物抑制剂-1活性的变化。
Int J Clin Lab Res. 2000;30(4):179-85. doi: 10.1007/s005990070004.
7
[The C1166 allele of the AT1R gene associated with ACE DD phenotype increases the risk for deep venous thrombosis].
Minerva Cardioangiol. 1999 Dec;47(12):530.
8
Recurrent in-stent restenosis is not associated with the angiotensin-converting enzyme D/I, angiotensinogen Thr174Met and Met235Thr, and the angiotensin-II receptor 1 A1166C polymorphism.支架内再狭窄的复发与血管紧张素转换酶D/I、血管紧张素原Thr174Met和Met235Thr以及血管紧张素II 1型受体A1166C多态性无关。
J Invasive Cardiol. 2007 Jun;19(6):261-4.
9
Restenosis after percutaneous coronary intervention is associated with the angiotensin-II type-1 receptor 1166A/C polymorphism but not with polymorphisms of angiotensin-converting enzyme, angiotensin-II receptor, angiotensinogen or heme oxygenase-1.经皮冠状动脉介入治疗后的再狭窄与血管紧张素 II 1 型受体 1166A/C 多态性相关，但与血管紧张素转换酶、血管紧张素 II 受体、血管紧张素原或血红素加氧酶-1 的多态性无关。
Pharmacogenet Genomics. 2006 May;16(5):331-7. doi: 10.1097/01.fpc.0000205001.07054.fa.
10
Influence of angiotensinogen M253T gene polymorphism and an angiotensin converting enzyme inhibitor on restenosis after percutaneous coronary intervention.血管紧张素原M253T基因多态性及血管紧张素转换酶抑制剂对经皮冠状动脉介入术后再狭窄的影响。
Atherosclerosis. 2002 Feb;160(2):339-44. doi: 10.1016/s0021-9150(01)00592-5.

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Pathogenesis and Clinical Significance of In-Stent Restenosis in Patients with Diabetes.糖尿病患者支架内再狭窄的发病机制及临床意义。
Int J Environ Res Public Health. 2021 Nov 15;18(22):11970. doi: 10.3390/ijerph182211970.
2
Influence of the angiotensin converting enzyme insertion or deletion genetic variant and coronary restenosis risk: evidence based on 11,193 subjects.血管紧张素转换酶插入或缺失基因变异与冠状动脉再狭窄风险的关系：基于11193名受试者的证据
PLoS One. 2013 Dec 13;8(12):e83415. doi: 10.1371/journal.pone.0083415. eCollection 2013.
3
Genetic polymorphism of angiotensin converting enzyme and risk of coronary restenosis after percutaneous transluminal coronary angioplasties: evidence from 33 cohort studies.
血管紧张素转换酶基因多态性与经皮腔内冠状动脉成形术后冠状动脉再狭窄的风险：来自 33 项队列研究的证据。
PLoS One. 2013 Sep 30;8(9):e75285. doi: 10.1371/journal.pone.0075285. eCollection 2013.
4
Angiotensin converting enzyme insertion or deletion polymorphism and coronary restenosis: meta-analysis of 16 studies.血管紧张素转换酶插入/缺失多态性与冠状动脉再狭窄：16项研究的荟萃分析
BMJ. 2002 Sep 7;325(7363):517-20. doi: 10.1136/bmj.325.7363.517.